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    2C-T-4 molecular structure

    2C-T-4 Stats & Data

    T-4 2ct4 2c-t4 2-ct4 2ct-4
    NPS DataHub
    MW291.84
    FormulaC13H22ClNO2S
    CAS868738-44-7
    IUPAChydrogen 2-[2,5-dimethoxy-4-(propan-2-ylsulfanyl)phenyl]ethan-1-amine chloride
    SMILES[Cl-].NCCc1cc(OC)c(SC(C)C)cc1OC.[H+]
    InChIKeyZKVCWPPXXYVULN-UHFFFAOYSA-N
    Phenethylamines; 2020/1. Von 2-Phenethylamin abgeleitete Verbindungen; 2021/1. Von 2-Phenethylamin abgeleitete Verbindungen; 2022/1. Von 2-Phenethylamin abgeleitete Verbindungen
    Chemical Class Phenethylamine
    Psychoactive Class Psychedelic
    Half-Life Unknown in humans; effect duration suggests a multi‑hour active window but pharmacokinetics have not been formally characterized.

    Receptor Profile

    Receptor Actions

    Agonists
    5-HT2A receptor agonist (partial)
    5-HT2B receptor agonist
    5-HT2C receptor agonist

    History & Culture

    2C-T-4 was first documented in the scientific literature by Alexander Shulgin and his colleagues in a 1991 journal article. Later that same year, Shulgin provided a more comprehensive account of the compound in his book PiHKAL (Phenethylamines I Have Known and Loved), where he devoted a chapter to describing his personal experiences with the substance. In these writings, Shulgin reported an intense "plus-four" experience on his rating scale at a dose of 12 milligrams, representing the highest possible classification for subjective psychedelic effects. The abbreviated name "T-4" has occasionally been used to refer to 2C-T-4, though this terminology carries potential for confusion. The same designation is shared by cyclotrimethylenetrinitramine (RDX), a powerful explosive, as well as thyroxine, a naturally occurring amino acid hormone in the body. Shulgin himself noted this naming overlap in his writings, clarifying that the psychedelic compound shares nothing beyond the shorthand with either of these unrelated substances.

    Effect Profile

    Curated + 20 Reports
    Psychedelic 8.8

    Strong visuals, headspace, auditory effects, and body load

    Visual Intensity×3
    1010
    Headspace Depth×3
    1010
    Auditory Effects×1
    1010
    Body Load / Somatic Effects×1
    1010
    Catalog Erowid

    Duration Timeline

    Bluelight
    Onset Comeup Peak Offset After Effects
    Oral
    30 minutes - 2.0 hours
    1-2 hours
    3-6 hours
    4-6 hours
    4-8 hours
    Total: 10-18 hours
    Insufflated
    4-19 minutes
    30 minutes - 1.0 hours
    3-6 hours
    4-6 hours
    4-8 hours
    Total: 10-18 hours

    Tolerance & Pharmacokinetics

    drugs.wiki
    Half-Life
    Unknown in humans; effect duration suggests a multi‑hour active window but pharmacokinetics have not been formally characterized.
    Addiction Potential
    Very low. No evidence of physical dependence; psychological habituation possible with frequent use, similar to other classical psychedelics.

    Tolerance Decay

    Full tolerance 1d Half tolerance 7d Baseline ~14d

    Model extrapolated from serotonergic psychedelic tolerance patterns; individual variability high; data are largely anecdotal for 2C‑T‑4 specifically.

    Cross-Tolerances

    LSD
    50% ●○○
    Psilocybin
    50% ●○○
    Other 2C phenethylamines
    60% ●●○

    Experience Report Analysis

    Erowid
    20 Reports
    2003–2022 Date Range
    5 With Age Data
    25 Effects Detected

    Demographics

    Gender Distribution

    Age Distribution

    Reports Over Time

    Effect Analysis

    Erowid

    Effects aggregated from 20 experience reports (20 Erowid)

    20 Reports
    25 Effects Detected
    9 Positive
    8 Adverse
    8 Neutral

    Effect Sentiment Distribution

    Confidence Distribution

    Positive Effects 9

    Color Enhancement 65.0% 70%
    Music Enhancement 50.0% 70%
    Stimulation 50.0% 70%
    Tactile Enhancement 40.0% 70%
    Empathy 40.0% 70%
    Euphoria 35.0% 70%
    Introspection 30.0% 70%
    Focus Enhancement 30.0% 70%
    Body High 25.0% 70%

    Adverse Effects 8

    Nausea 50.0% 70%
    Muscle Tension 40.0% 70%
    Anxiety 40.0% 70%
    Confusion 25.0% 70%
    Pupil Dilation 25.0% 70%
    Headache 20.0% 70%
    Sweating 15.0% 70%
    Increased Heart Rate 15.0% 70%

    Dosage Distribution

    Dose distribution from experience reports

    Median: 15.0 mg IQR: 10.0–16.0 mg n=14

    Real-World Dose Distribution

    62K Doses

    From 31 individual dose entries

    Oral (n=29)

    Median: 15.0mg 25th: 8.0mg 75th: 18.0mg 90th: 22.0mg
    mg/kg median: 0.183 mg/kg 75th: 0.277

    Form / Preparation

    Most common forms and preparations reported

    Body-Weight Dosing

    Dose relative to body weight from reports with weight data

    Median: 0.189 mg/kg IQR: 0.178–0.268 mg/kg n=13

    Redose Patterns

    Redosing behavior across 16 reports

    18.8% Redosed
    1.3 Avg Doses
    60m Median Interval

    Legal Status

    Country Status Notes
    Canada Schedule III (CDSA) Designated as a controlled substance under Schedule III of the Controlled Drugs and Substances Act effective October 31, 2016. Possession, trafficking, and production carry criminal penalties.
    China Controlled substance Added to the national list of controlled substances in October 2015. Manufacturing, distribution, and possession are prohibited.
    Denmark Schedule B Listed as a Schedule B controlled substance under Danish drug control legislation. This classification restricts possession and distribution.
    Sweden Prohibited (health hazard) Classified as a health hazard by the Swedish National Institute of Public Health (Statens folkhälsoinstitut) on July 15, 2007 under regulation SFS 2007:600. Controlled under the Act on the Prohibition of Certain Goods Dangerous to Health (Lagen om förbud mot vissa hälsofarliga varor), making both sale and possession illegal.
    United States Schedule I Designated as a Schedule I controlled substance on July 9, 2012 under the Synthetic Drug Abuse Prevention Act of 2012. Classified as having high abuse potential with no currently accepted medical use in treatment.

    Harm Reduction

    drugs.wiki

    • Identity and isomer check: historic market confusion between 2C‑T‑4 and the inactive isomer psi‑2C‑T‑4 existed due to a gamma/psi transcription error in early online PiHKAL copies; ensure proper labeling and avoid vendors using “gamma‑2C‑T‑4.”

    • Reagent tests: Marquis typically shifts orange→salmon/red with 2C‑T‑4, similar to other 2C‑T compounds; reagent colors cannot distinguish 2C‑T‑2/‑4/‑7, so laboratory verification is recommended.

    • Come‑up can be slow/uneven; avoid redosing for at least 3 hours. Related thio‑phenethylamines (e.g., 2C‑T‑7) frequently take 1–3 h to fully develop orally.

    • Insufflation significantly increases adverse effects with this family and has been implicated in multiple 2C‑T‑7 fatalities; oral administration is the lower‑risk route.

    • Expect moderate body load (nausea, muscle tension, vasoconstriction) and difficulty sleeping; plan a next‑day recovery window and avoid late‑evening dosing.

    • Tolerance builds acutely after one session and typically halves over ~7 days, with cross‑tolerance to other serotonergic psychedelics (LSD, psilocybin, 2C‑x).

    • Avoid combinations with lithium, MAOIs, tramadol, or strong stimulants. Cannabis can unpredictably intensify effects.

    • Market adulteration/misrepresentation: materials sold as “2C” or “tusi/pink cocaine” often contain mixed stimulants, ketamine, MDxx, or trace opioids rather than true 2C compounds; use trusted lab checking services when possible.

    • Use a 0.001 g (1 mg) scale; the dose‑response becomes steep beyond ~15 mg in many users, and inter‑individual variability is high.

    • Eat lightly 3–4 h prior and sip fluids; antiemetics like ondansetron (non‑sedating, non‑serotonergic) have been used anecdotally for nausea but avoid serotonergic agents.

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