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    PCP molecular structure

    PCP Stats & Data

    Wet Dust Boat Zoom Sherm angeldust phencyclidine angel
    NPS DataHub
    MW243.39
    FormulaC17H25N
    CAS77-10-1
    IUPAC1-(1-Phenylcyclohexyl)piperidine
    SMILESC1CCN(CC1)C1(CCCCC1)c1ccccc1
    InChIKeyJTJMJGYZQZDUJJ-UHFFFAOYSA-N
    2021/6. Von Arylcyclohexylamin abgeleitete Verbindungen; 2022/6. Von Arylcyclohexylamin abgeleitete Verbindungen
    Psychoactive Class Dissociative / Psychedelic
    Half-Life ~21 hours typical; published ranges ~7–50 hours with prolonged symptoms due to redistribution from fat tissue.

    Interaction Warnings

    psychedelics

    This combination is not advised because PCP has been reported to cause extreme psychological disturbances such as psychosis and mania at a significantly higher rate than other dissociatives.

    stimulants

    This combination is not advised because PCP has been reported to cause extreme psychological disturbances such as psychosis and mania at a significantly higher rate than other dissociatives.

    Pharmacology

    DrugBank
    State Solid

    Description

    A hallucinogen formerly used as a veterinary anesthetic, and briefly as a general anesthetic for humans. Phencyclidine is similar to ketamine in structure and in many of its effects. Like ketamine, it can produce a dissociative state. It exerts its pharmacological action through inhibition of NMDA receptors (receptors, N-methyl-D-aspartate). As a drug of abuse, it is known as PCP and Angel Dust.

    Mechanism of Action

    The N-methyl-D-Aspartate (NMDA) receptor, a type of ionotropic receptor, is found on the dendrites of neurons and receives signals in the form of neurotransmitters. It is a major excitatory receptor in the brain. Normal physiological function requires that the activated receptor fluxes positive ions through the channel part of the receptor. PCP enters the ion channel from the outside of the neuron and binds, reversibly, to a site in the channel pore, blocking the flux of positive ions into the cell. PCP therefore inhibits depolarization of neurons and interferes with cognitive and other functions of the nervous system.

    Pharmacodynamics

    Phencyclidine works primarily as an NMDA receptor antagonist, which blocks the activity of the NMDA Receptor.

    Receptor Profile

    Receptor Actions

    Agonists
    Sigma-2 receptor agonist
    Dopamine D2 receptor agonist
    Antagonists
    NMDA receptor antagonist (noncompetitive)
    Inhibitors
    Serotonin reuptake inhibitor (mild)

    History & Culture

    1926–1978

    Phencyclidine was first synthesized in 1926 by German chemist Arthur Kötz and his student Paul Merkel during research involving Grignard reactions with piperidinocyclohexancarbonitrile compounds. The compound remained largely unexplored until 1956, when chemist H. Victor Maddox independently synthesized it and the pharmaceutical company Parke-Davis (now part of Pfizer) developed it as an anesthetic medication. The drug's clinical career proved brief. By 1965, medical use in humans was prohibited in the United States due to an unacceptably high rate of adverse effects in patients. Veterinary applications continued for over a decade longer, but these too were discontinued in 1978. The development of ketamine, which produced similar anesthetic effects with better tolerability, rendered PCP obsolete in legitimate medicine.

    1960–1990

    While still technically available as a medical anesthetic, PCP began appearing as a street drug in major American cities during the 1960s. Its recreational use grew substantially over the following decade, reaching a peak in the late 1970s. By 1978, prominent media outlets including People magazine and the CBS program 60 Minutes had declared it the nation's most pressing drug concern. The trajectory reversed sharply in the following decade. Surveys of American high school students showed that 13% reported having tried PCP at least once in 1979; by 1990, this figure had dropped below 3%. Despite this overall decline, the substance has remained in circulation, particularly in certain American urban areas. Emergency department visits related to PCP increased between 2005 and 2011, and as of 2022, approximately 0.7% of American twelfth-grade students reported use within the prior year.

    PCP developed a fearsome popular reputation centered on claims that users became extraordinarily violent, impervious to pain, and capable of superhuman physical feats. Stories of users pulling out their own teeth, destroying property, and attacking others with unusual aggression became fixtures of drug scare coverage. Research conducted by the Drug Abuse Warning Network during the 1970s found these media accounts to be substantially exaggerated. Studies indicated that violent incidents were uncommon and typically involved individuals with pre-existing patterns of aggressive behavior regardless of substance use. Notably, the dramatic violent reactions described in news coverage were not observed during the drug's legitimate clinical use as an anesthetic in the 1950s and 1960s, and subsequent investigations have frequently found reports of PCP-induced violence to lack evidentiary foundation.

    The drug's notoriety during its peak usage period generated various cultural representations. American artist Jean-Michel Basquiat referenced the substance in his 1982 painting "Dustheads," which depicted two individuals using angel dust. The Japanese manga series City Hunter, created by Tsukasa Hojo in 1985, featured a drug called "Angel Dust," though a 2023 animated film adaptation reimagined this as a fictional nanomachine serum rather than the actual substance. The animated series Hazbin Hotel features a character who adopted "Angel Dust" as his chosen name.

    Effect Profile

    Curated + 132 Reports
    Psychedelic 8.4

    Strong visuals and auditory effects with moderate headspace, mild body load

    Visual Intensity×3
    109.02.0
    Headspace Depth×3
    73.41.9
    Auditory Effects×1
    10101.8
    Body Load / Somatic Effects×1
    42.02.7
    Catalog Erowid BlueLight
    Dissociative 6.9

    Strong dissociative depth, mania, motor impairment, and insight

    Dissociative Depth×3
    105.95.6
    Mania / Compulsion×1
    103.61.4
    Insight / Novel Thought×2
    93.2
    Motor / Sensory Impairment×1
    104.01.8
    Catalog Erowid BlueLight

    Duration Timeline

    Bluelight
    Onset Comeup Peak Offset After Effects
    Oral
    30 minutes - 1.5 hours
    42 minutes - 2.0 hours
    2-3 hours
    1-2 hours
    4-48 hours
    Total: 4-6 hours
    Smoked
    1-4 minutes
    15-30 minutes
    2-4 hours
    2-3 hours
    24-48 hours
    Total: 4-6 hours
    Insufflated
    6-30 minutes
    30 minutes - 1.5 hours
    2-3 hours
    1-2 hours
    4-48 hours
    Total: 4-6 hours

    Community Effects

    TripSit
    Positive
    euphoria dissociation stimulation analgesia
    Negative
    mania psychosis confusion amnesia seizures addiction

    Tolerance & Pharmacokinetics

    drugs.wiki
    Half-Life
    ~21 hours typical; published ranges ~7–50 hours with prolonged symptoms due to redistribution from fat tissue.
    Addiction Potential
    Moderate to high psychological dependence risk. Compulsive patterns and craving are reported with repeated use; chronic intake is associated with persistent cognitive/speech problems and depressive states in some users.

    Tolerance Decay

    Full tolerance 3d Half tolerance 7d Baseline ~21d

    Rapid tolerance develops with repeated exposure and partially generalizes across NMDA-antagonist dissociatives. Anecdotal and clinical observations suggest tolerance recedes substantially over 2–4 weeks of abstinence; prolonged heavy use may leave residual cognitive changes beyond simple tolerance. Data quality is limited and varies by compound and route.

    Cross-Tolerances

    Ketamine
    50% ●○○
    DXM
    40% ●○○
    3-MeO-PCP and related arylcyclohexylamines
    60% ●●○

    Experience Report Analysis

    Erowid BlueLight
    104 Reports
    1992–2017 Date Range
    19 With Age Data
    28 Effects Detected

    Demographics

    Gender Distribution

    Age Distribution

    Reports Over Time

    Effect Analysis

    Erowid + Bluelight

    Effects aggregated from 132 experience reports (104 Erowid + 28 Bluelight)

    132 Reports
    125 Effects Detected
    47 Positive
    51 Adverse
    27 Neutral

    Effect Sentiment Distribution

    Confidence Distribution

    Positive Effects 47

    Euphoria 24.2% 89%
    Music Enhancement 21.2% 86%
    Stimulation 18.9% 82%
    Contentment 17.9% 82%
    Color Enhancement 15.9% 82%
    Body High 13.6% 83%
    Tactile Enhancement 13.5% 70%
    Focus Enhancement 12.1% 80%
    Pain Suppression 10.7% 80%
    Geometric Imagery 10.7% 78%
    Environmental Transfiguration 10.7% 78%
    Empathogenic Connection 10.7% 78%
    Sociability Enhancement 10.7% 83%
    Libido Enhancement 10.7% 82%
    Empathy 10.6% 82%
    Introspection 8.3% 84%
    Ego Inflation 7.1% 82%
    Surface Breathing 7.1% 80%
    Emotional Openness 7.1% 85%
    Visual Vibration 7.1% 75%

    Adverse Effects 51

    Anxiety 39.4% 80%
    Confusion 30.3% 88%
    Depersonalization 21.4% 84%
    Amnesia 17.9% 83%
    Heaviness 17.9% 81%
    Fear 17.9% 88%
    Motor Impairment 15.9% 85%
    Delusion 14.3% 85%
    Memory Suppression 12.9% 83%
    Paranoia 10.7% 87%
    Ataxia 10.7% 85%
    Vomiting 10.7% 83%
    Tremor 10.7% 78%
    Dysphoria 10.7% 83%
    Panic 10.7% 90%
    Nausea 9.1% 84%
    Sweating 8.3% 81%
    Psychosis 7.7% 70%
    Body Temperature Change 7.1% 75%
    Focus Suppression 7.1% 85%

    Real-World Dose Distribution

    62K Doses

    From 45 individual dose entries

    Common Combinations

    Most co-occurring substances in experience reports

    Form / Preparation

    Most common forms and preparations reported

    Redose Patterns

    Redosing behavior across 51 reports

    7.8% Redosed
    1.1 Avg Doses

    Harm Reduction

    drugs.wiki

    PCP is highly lipophilic; symptoms can recur in waves hours to days later as drug redistributes from fat — avoid redosing to chase effects. Heat injury is a major risk during agitation or restraint; rapid cooling and prompt medical help are lifesaving in severe hyperthermia. Analgesia and dissociation mask injuries — avoid heights, water, stoves, traffic, and never drive for at least a full day after strong effects. Smoked “wet/sherm” is usually PCP solution, not real embalming fluid; formaldehyde is carcinogenic and should not be inhaled. Street strengths vary widely; use milligram-accurate scales and avoid eyeballing crystals or pre-dipped cigarettes. Combination with depressants (alcohol, benzos, opioids, GHB) greatly increases blackout, airway, and aspiration risks; medical teams use benzodiazepines for agitation and seizures — this is not a justification to self-mix. Stimulant co-use (cocaine, amphetamines) raises risks of psychosis, hyperthermia, and rhabdomyolysis. False positives for PCP occur on immunoassay urine screens (e.g., from DXM, venlafaxine, tramadol); confirmatory GC/MS is more specific. If severe agitation, chest pain, seizures, or very high temperature occur, call emergency services; quiet, low-stimulation environments reduce escalation. Consider drug checking where available (FTIR/GC–MS) as PCP may be mis-sold or cut with other arylcyclohexylamines; pre-dipped cigarettes are especially unpredictable. Frequent use builds strong tolerance and can lead to prolonged cognitive and mood disturbances; spacing use by several weeks reduces risk.

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