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    3-HO-PCP molecular structure

    3-HO-PCP Stats & Data

    3-oh-pcp Hydroxyphencyclidine 3hopcp
    NPS DataHub
    MW259.39
    FormulaC17H25NO
    CAS79787-43-2
    IUPAC3-(1-piperidin-1-ylcyclohexyl)phenol
    SMILESOc1cccc(c1)C1(CCCCC1)N1CCCCC1
    InChIKeyAMSXTZUCNOKUEN-UHFFFAOYSA-N
    Arylcyclohexylamines; 2021/6. Von Arylcyclohexylamin abgeleitete Verbindungen; 2022/6. Von Arylcyclohexylamin abgeleitete Verbindungen
    Psychoactive Class Dissociative
    Half-Life Unknown in humans; acute effects suggest hours‑scale persistence with multi‑hour after‑effects.

    Receptor Profile

    Receptor Actions

    Agonists
    μ-opioid receptor agonist (partial)
    κ-opioid receptor agonist
    Sigma-1 receptor agonist
    Antagonists
    NMDA receptor antagonist (noncompetitive)

    Receptor Binding

    NMDA antagonist

    History & Culture

    3-HO-PCP was first synthesized in 1978 during investigations into the structure-activity relationships of phencyclidine (PCP) derivatives. Throughout the 1980s, researchers continued to characterize its pharmacological properties, discovering its activity at opioid receptors in animal models during this period. The compound remained largely unknown outside academic circles until 1999, when a chemist operating under the pseudonym "John Q. Beagle" discussed it in a post on The Hive, a now-defunct online chemistry forum. Beagle highlighted the compound's substantially increased potency compared to PCP and its notable affinity for opioid receptors. Even before the substance became commercially available, members of the Bluelight forum had begun documenting its psychoactive effects based on personal experimentation. Around 2009, 3-HO-PCP entered the research chemical market through online vendors. Despite being structurally related to PCP—a compound that had developed considerable notoriety through decades of media coverage and street names like "angel dust," "rocket fuel," and "sherm"—3-HO-PCP has remained one of the more obscure members of the arylcyclohexylamine class. It continues to be distributed primarily through gray-market online sources, with very little formal research having been conducted on its effects, metabolism, or toxicity in humans.

    Effect Profile

    Curated + 22 Reports
    Dissociative 6.6

    Strong dissociative depth, motor impairment, and mania with moderate insight

    Dissociative Depth×3
    1010
    Mania / Compulsion×1
    8
    Insight / Novel Thought×2
    78.0
    Motor / Sensory Impairment×1
    10
    Catalog BlueLight

    Duration Timeline

    Bluelight
    Onset Comeup Peak Offset After Effects
    Oral
    15-45 minutes
    1-1.5 hours
    2-4 hours
    1-2 hours
    12 hours
    Total: 3-4 hours
    Insufflated
    4-19 minutes
    30 minutes - 1.0 hours
    2-3 hours
    1-2 hours
    12 hours
    Total: 3-4 hours

    Community Effects

    TripSit
    Positive
    stimulation dissociation
    Negative
    nausea tachycardia muscle tension seizures

    Tolerance & Pharmacokinetics

    drugs.wiki
    Half-Life
    Unknown in humans; acute effects suggest hours‑scale persistence with multi‑hour after‑effects.
    Addiction Potential
    Non-medical use shows a notable tendency toward compulsive redosing and binge patterns similar to other PCP-like dissociatives. Frequent/high dosing can lead to tolerance escalation, blackouts, and functional impairment; many users report difficulty moderating use once intoxicated.

    Tolerance Decay

    Full tolerance 2d Half tolerance 3d Baseline ~14d

    Tolerance builds rapidly with repeated use over days and decays over 1–2+ weeks; numbers are approximate and largely anecdotal for this specific analogue.

    Cross-Tolerances

    Ketamine
    60% ●○○
    3‑MeO‑PCP
    70% ●○○
    O‑PCE/3‑HO‑PCE
    60% ●○○

    Experience Report Analysis

    Erowid BlueLight
    17 Reports
    2018–2023 Date Range
    17 With Age Data
    23 Effects Detected

    Demographics

    Gender Distribution

    Age Distribution

    Reports Over Time

    Effect Analysis

    Erowid + Bluelight

    Effects aggregated from 22 experience reports (17 Erowid + 5 Bluelight)

    22 Reports
    58 Effects Detected
    20 Positive
    24 Adverse
    14 Neutral

    Effect Sentiment Distribution

    Confidence Distribution

    Positive Effects 20

    Color Enhancement 52.9% 70%
    Music Enhancement 52.9% 70%
    Tactile Enhancement 47.1% 70%
    Stimulation 45.5% 80%
    Euphoria 40.9% 90%
    Introspection 40.0% 82%
    Focus Enhancement 35.3% 70%
    Empathy 35.3% 70%
    Emotional Openness 20.0% 80%
    Tinnitus 20.0% 90%
    Joy 20.0% 85%
    Mystical Quality 20.0% 90%
    Revelatory Insight 20.0% 85%
    Oneness 20.0% 85%
    Gratitude 20.0% 85%
    Peace 20.0% 90%
    Contentment 20.0% 85%
    Empathogenic Connection 20.0% 80%
    Insight 20.0% 90%
    Pain Relief 17.6% 70%

    Adverse Effects 24

    Fear 60.0% 87%
    Confusion 45.5% 92%
    Anxiety 45.4% 90%
    Motor Impairment 40.9% 88%
    Vomiting 40.0% 85%
    Memory Suppression 31.8% 85%
    Psychosis 23.5% 70%
    Amnesia 20.0% 95%
    Dry Mouth 20.0% 90%
    Involuntary Movements 20.0% 85%
    Numbness 20.0% 95%
    Dizziness 20.0% 90%
    Blurred Vision 20.0% 85%
    Chills 20.0% 85%
    Body Temperature Change 20.0% 80%
    Headache 20.0% 85%
    Pressure 20.0% 90%
    Jaw Clenching 20.0% 75%
    Delusion 20.0% 95%
    Panic 20.0% 90%

    Real-World Dose Distribution

    62K Doses

    From 160 individual dose entries

    Oral (n=71)

    Median: 11.0mg 25th: 10.0mg 75th: 15.0mg 90th: 20.0mg
    mg/kg median: 0.038 mg/kg 75th: 0.08

    Rectal (n=69)

    Median: 15.0mg 25th: 11.0mg 75th: 15.0mg 90th: 20.0mg

    Topical (n=5)

    Median: 1.0mg 25th: 1.0mg 75th: 1.0mg 90th: 1.0mg

    Insufflated (n=11)

    Median: 10.0mg 25th: 10.0mg 75th: 13.5mg 90th: 18.0mg
    mg/kg median: 0.147 mg/kg 75th: 0.193

    Form / Preparation

    Most common forms and preparations reported

    Body-Weight Dosing

    Dose relative to body weight from reports with weight data

    Median: 0.182 mg/kg IQR: 0.147–0.203 mg/kg n=6

    Redose Patterns

    Redosing behavior across 13 reports

    46.2% Redosed
    1.9 Avg Doses
    75m Median Interval

    Legal Status

    Country Status Notes
    Czech Republic Schedule I (List 4) Controlled under Nařízení vlády č. 463/2013 Sb. (§ 1, d), 1.). Permitted for research purposes and restricted therapeutic applications only.
    Germany Unscheduled Not listed as a controlled substance under the Betäubungsmittelgesetz (BtMG). However, under §2 of the Arzneimittelgesetz (AMG), sale for human consumption remains prohibited.
    Sweden Illegal Prohibited substance under Swedish drug control legislation.
    Switzerland Controlled (Verzeichnis E) Specifically named as a controlled substance in Verzeichnis E of Swiss narcotics legislation.
    Turkey Illegal Classified as a controlled drug under Turkish law. Possession, production, supply, and importation are prohibited.
    United Kingdom Class B Controlled under the Misuse of Drugs Act 1971 via the arylcyclohexylamine generic clause introduced by S.I. 2013/239, effective February 26, 2013. As a derivative of 1-phenylcyclohexylamine with a 1-piperidyl group and hydroxy substitution on the phenyl ring, it falls within this catch-all provision. Possession, production, supply, and importation are criminal offenses.

    Harm Reduction

    drugs.wiki

    Potency is high and interindividual variability is large; 1–2 mg can noticeably change effects, so weigh with a calibrated 0.001 g scale or volumetrically. Onset—especially intranasal—can be delayed 40–90+ minutes; redosing during the come‑up is a common trigger for blackouts and hazardous behavior. Several experienced users and moderators note unusually prominent muscle tension/soreness after higher doses; spacing sessions reduces cumulative strain. Intranasal use is often caustic and can inflame sinuses; oral routes are gentler on mucosa. Mislabeling/adulteration has been documented in the wild (e.g., MD‑PiHP sold as 3‑HO‑PCP); use professional drug checking when possible, and do not assume reagent tests can definitively identify PCP analogues. The compound’s opioid receptor profile in vitro is debated; regardless, mixing with CNS depressants increases sedation and slows breathing—avoid these combinations. Dissociatives in general can aggravate mood instability, mania, and psychosis; people with a history of these should avoid or use only with strong support and conservative dosing in a safe setting. Heavy, frequent dissociative use (well‑established for ketamine) is linked to urinary tract problems; while specific data for 3‑HO‑PCP are limited, adopt the same precautions—hydrate, avoid daily use, and stop if urinary symptoms appear. Because ambulation can be preserved while judgment is impaired, trips should be done sitting/lying down with a sober sitter available to prevent accidents.

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