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    3-MeO-PCP molecular structure

    3-MeO-PCP Stats & Data

    3-meo 3meopcp
    NPS DataHub
    MW309.88
    FormulaC18H28ClNO
    CAS91164-58-8
    IUPAC1-1-3-methoxyphenyl cyclohexyl -piperidine
    SMILES[Cl-].COc1cccc(c1)C1(CCCCC1)N1CCCCC1.[H+]
    InChIKeyMFBXZMQWSUHTBV-UHFFFAOYSA-N
    2021/6. Von Arylcyclohexylamin abgeleitete Verbindungen; 2022/6. Von Arylcyclohexylamin abgeleitete Verbindungen
    Psychoactive Class Dissociative
    Half-Life Estimated several hours; reports commonly cite 4–8 h elimination half‑life with prolonged after‑effects.

    Interaction Warnings

    psychedelics

    This combination is not advised because 3-MeO-PCP has been reported to cause extreme psychological disturbances such as psychosis and mania at a significantly higher rate than other dissociatives.

    stimulants

    This combination is not advised because 3-MeO-PCP has been reported to cause extreme psychological disturbances such as psychosis and mania at a significantly higher rate than other dissociatives.

    Receptor Profile

    Receptor Actions

    Agonists
    Sigma-1 receptor agonist (Ki 42 nM)
    Antagonists
    NMDA receptor antagonist (Ki 20 nM)
    Inhibitors
    Serotonin reuptake inhibitor (SERT Ki 216 nM)

    Receptor Binding

    NMDA antagonist

    History & Culture

    1979–present

    3-MeO-PCP was first synthesized in 1979 by Geneste and colleagues during an investigation into phencyclidine derivatives. This work followed earlier research by Maddox et al. in 1965, which had produced the related compounds 2-MeO-PCP and 4-MeO-PCP as part of an exploration into potential central nervous system depressants. Following its synthesis, 3-MeO-PCP remained largely confined to occasional scientific research for approximately two decades. During this period, researchers such as Vignon et al. in 1982 conducted investigations into its pharmacological properties, but its activity in humans remained undocumented in the scientific literature.

    1999–present

    A chemist operating under the pseudonym John Q. Beagle provided the first documented description of 3-MeO-PCP's effects in humans in 1999. Posting on The Hive, an online drug chemistry forum, Beagle reported that the compound produced effects qualitatively similar to PCP with comparable potency. Despite this early report, the substance did not gain significant attention for another decade.

    2009–2015

    Discussion of 3-MeO-PCP began appearing on drug information forums around 2009, with one of the earliest reports coming from a user named 'hugo24' on Bluelight.org. By April 2011, online vendors including Buy Research Chemicals UK had begun selling the compound, and the number of available sources increased alongside a growing collection of user experience reports, many of which were positive in tone. Analysis of forum activity data from Drugs-Forum.com revealed that 3-MeO-PCP first appeared around 2011, reached peak discussion levels between 2011 and 2013, and had largely faded from active discussion by early 2016. The substance was first reported to the European Union's Early Warning System in March 2012 by the United Kingdom. Sweden's STRIDA project, which monitors suspected intoxications involving novel psychoactive substances, first detected 3-MeO-PCP in the fourth quarter of 2013. Fatalities associated with 3-MeO-PCP began appearing by the mid-2010s. In October 2014, the European Monitoring Centre for Drugs and Drug Addiction received notification of three deaths linked to the substance in Sweden, representing some of the earliest documented fatalities. These reports contributed to regulatory responses across multiple jurisdictions, with Sweden implementing controls in early 2015 and several other European countries following with scheduling measures throughout 2015 and subsequent years.

    Effect Profile

    Curated + 62 Reports
    Dissociative 7.1

    Strong dissociative depth, mania, insight, and motor impairment

    Dissociative Depth×3
    10105.1
    Mania / Compulsion×1
    103.8
    Insight / Novel Thought×2
    108.0
    Motor / Sensory Impairment×1
    106.31.3
    Catalog Erowid BlueLight

    Duration Timeline

    Bluelight
    Onset Comeup Peak Offset After Effects
    Oral
    30 minutes - 1.5 hours
    45 minutes - 2.0 hours
    2-3 hours
    1-2 hours
    4-48 hours
    Total: 3-5 hours
    Insufflated
    4-30 minutes
    45 minutes - 1.5 hours
    1.5-2 hours
    45 minutes - 1.0 hours
    4-48 hours
    Total: 2-4 hours
    Sublingual
    2-48 hours
    Smoked
    2-48 hours

    Community Effects

    TripSit
    Positive
    euphoria dissociation sedation
    Negative
    mania psychosis amnesia

    Tolerance & Pharmacokinetics

    drugs.wiki
    Half-Life
    Estimated several hours; reports commonly cite 4–8 h elimination half‑life with prolonged after‑effects.
    Addiction Potential
    Moderate to high psychological reinforcement; some users report compulsive redosing and mania with frequent or high‑dose use.

    Tolerance Decay

    Full tolerance 3d Half tolerance 7d Baseline ~14d

    Model reflects rapid tolerance build with frequent use and slow decay over weeks as commonly reported for arylcyclohexylamines; confidence is limited due to reliance on user reports rather than controlled studies.

    Cross-Tolerances

    Ketamine
    50% ●○○
    PCP
    60% ●○○
    Other arylcyclohexylamines (e.g., 3‑MeO‑PCE, 3‑HO‑PCP)
    60% ●○○

    Experience Report Analysis

    Erowid BlueLight
    47 Reports
    2011–2021 Date Range
    46 With Age Data
    29 Effects Detected

    Demographics

    Gender Distribution

    Age Distribution

    Reports Over Time

    Effect Analysis

    Erowid + Bluelight

    Effects aggregated from 62 experience reports (47 Erowid + 15 Bluelight)

    62 Reports
    94 Effects Detected
    35 Positive
    38 Adverse
    21 Neutral

    Effect Sentiment Distribution

    Confidence Distribution

    Positive Effects 35

    Euphoria 45.2% 86%
    Music Enhancement 45.1% 95%
    Stimulation 45.1% 80%
    Empathy 42.6% 70%
    Tactile Enhancement 42.0% 80%
    Color Enhancement 42.0% 88%
    Joy 33.3% 84%
    Focus Enhancement 30.7% 80%
    Contentment 26.7% 81%
    Sociability Enhancement 26.7% 81%
    Introspection 25.8% 81%
    Body High 24.2% 85%
    Awe 20.0% 75%
    Insight 20.0% 85%
    Peace 13.3% 82%
    Pain Relief 11.3% 88%
    Visual Trails 6.7% 85%
    Brightness Enhancement 6.7% 85%
    Thought Connectivity 6.7% 70%
    Drowsiness 6.7% 75%

    Adverse Effects 38

    Anxiety 38.8% 82%
    Confusion 30.7% 82%
    Motor Impairment 27.4% 82%
    Fear 26.7% 88%
    Muscle Tension 17.0% 70%
    Memory Suppression 17.0% 70%
    Nausea 14.9% 70%
    Delusion 13.3% 90%
    Panic 13.3% 88%
    Insomnia 13.3% 82%
    Communication Suppression 13.3% 82%
    Pupil Dilation 10.6% 70%
    Increased Heart Rate 10.6% 70%
    Restless Legs 6.7% 90%
    Itching 6.7% 85%
    Nystagmus 6.7% 85%
    Paranoid Ideation 6.7% 80%
    Entity Imagery 6.7% 75%
    Ego Inflation 6.7% 85%
    Cosmic Significance 6.7% 75%

    Dosage Distribution

    Dose distribution from experience reports

    Median: 12.0 mg IQR: 10.0–20.0 mg n=15

    Real-World Dose Distribution

    62K Doses

    From 73 individual dose entries

    Insufflated (n=38)

    Median: 14.5mg 25th: 7.0mg 75th: 19.75mg 90th: 25.0mg
    mg/kg median: 0.195 mg/kg 75th: 0.259

    Oral (n=12)

    Median: 8.0mg 25th: 6.0mg 75th: 10.0mg 90th: 10.0mg
    mg/kg median: 0.141 mg/kg 75th: 0.184

    Sublingual (n=8)

    Median: 10.0mg 25th: 10.0mg 75th: 10.75mg 90th: 13.6mg
    mg/kg median: 0.179 mg/kg 75th: 0.184

    Form / Preparation

    Most common forms and preparations reported

    Body-Weight Dosing

    Dose relative to body weight from reports with weight data

    Insufflated

    Median: 0.195 mg/kg IQR: 0.123–0.294 mg/kg n=15

    Oral

    Median: 0.147 mg/kg IQR: 0.097–0.431 mg/kg n=8

    Sublingual

    Median: 0.179 mg/kg IQR: 0.157–0.182 mg/kg n=5

    Redose Patterns

    Redosing behavior across 43 reports

    34.9% Redosed
    1.7 Avg Doses
    92m Median Interval

    Legal Status

    Country Status Notes
    Australia Prohibited (Schedule 9 analogue) Captured under broad drug analogue laws at both federal and state levels. As PCP is a Schedule 9 prohibited substance, 3-MeO-PCP is similarly prohibited as a structural analogue.
    Austria Illegal (NPSG) Controlled under the Neue-Psychoaktive-Substanzen-Gesetz (New Psychoactive Substances Act). Possession, production, and sale are prohibited.
    Brazil Controlled substance Added to Portaria SVS/MS nº 344 on May 17, 2018. Possession, distribution, and use are prohibited.
    Canada Schedule I (CDSA) Controlled under the Controlled Drugs and Substances Act as a PCP analogue. The CDSA places all PCP analogues, derivatives, and salts under Schedule I prohibition, making 3-MeO-PCP automatically banned despite not being mentioned by name.
    Chile Illegal Controlled under Ley 20000 (Ley de drogas). As an ether of PCP, 3-MeO-PCP falls under the prohibition of all PCP esters and ethers specified in Chilean drug law.
    Czech Republic Schedule I Became a scheduled substance effective October 1, 2015 under Government Decree no. 243/2015 Coll., which amended Government Regulation no. 463/2013 Coll.
    Denmark Illegal As of August 25, 2015, all MeO-PCP isomers including 3-MeO-PCP were added to the list of controlled substances.
    Germany Anlage II BtMG Added to Anlage II of the Betäubungsmittelgesetz on November 21, 2015. Production, sale, and possession are prohibited.
    Italy Schedule I Listed as a Schedule I controlled substance under Italian drug legislation. Possession, production, and distribution are prohibited.
    Netherlands Schedule I (Opiumwet) Listed as a Schedule I controlled substance under the Dutch Opium Act. Possession, production, and distribution are prohibited.
    Portugal Uncontrolled Not specifically controlled as 3-MeO-PCP is neither a salt nor an isomer of PCP under Portuguese drug legislation. Standard decriminalization policies for personal drug use may apply.
    Sweden Controlled substance The Swedish public health agency recommended classification as a hazardous substance on November 10, 2014. On January 16, 2015, 3-MeO-PCP was officially declared a controlled substance.
    Switzerland Controlled (Verzeichnis E) Specifically named as a controlled substance under Verzeichnis E of Swiss narcotics legislation.
    Turkey Illegal Classified as a controlled drug. Possession, production, supply, and importation are prohibited.
    United Kingdom Class B Controlled under the Misuse of Drugs Act 1971. Covered by the arylcyclohexylamine generic clause (S.I. 2013/239) which came into effect on February 26, 2013. This clause prohibits derivatives of 1-phenylcyclohexylamine where the amine has been replaced with a 1-piperidyl group and further substituted in the phenyl ring with an alkoxy substituent.
    United States Unscheduled (Federal Analogue Act applies) Not a federally scheduled substance. However, due to structural and pharmacological similarities to the Schedule II drug PCP, possession or distribution for human consumption could be prosecuted under the Federal Analogue Act. Virginia has specifically scheduled 3-MeO-PCP as a Schedule I substance under Code of Virginia 54.1-3446.

    Harm Reduction

    drugs.wiki

    3‑MeO‑PCP is active in the single‑milligram range; inaccurate scales can lead to large relative dosing errors, so volumetric dosing is strongly recommended. The dose–response curve is steep and onset can be delayed, making premature redosing a common cause of accidental over-intoxication. It is an NMDA receptor antagonist and has been described by TripSit as also exhibiting mild serotonin reuptake inhibition, so combinations with serotonergic substances warrant extra caution. Compared with ketamine, it can produce clearer stimulation but is also more likely to induce ambulatory delirium, disinhibition, and manic or psychotic states at strong doses. Erowid documents that at least one death has been associated with 3‑MeO‑PCP and warns of episodes where users cannot distinguish fantasy from reality, increasing risk of harm in uncontrolled environments. Avoid mixing with alcohol or benzodiazepines due to unpredictable sedation and accident risk; avoid driving or hazardous activities until the next day because after‑effects can persist up to 48 hours. People with a personal or family history of psychosis, bipolar disorder, or mania should avoid due to elevated risk of psychiatric destabilization. The RC market is prone to mislabeling and variable purity; reagent testing and conservative allergy tests reduce risk. Tolerance builds rapidly with frequent use; spacing sessions by multiple weeks reduces both tolerance and compulsive redosing patterns.

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