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    Clonidine molecular structure

    Clonidine Stats & Data

    Kapvay Dixarit Catapres Haemiton Duraclon
    Psychoactive Class Depressant

    Pharmacology

    DrugBank
    State Solid Clearance The clearance of clonidine is 1.9-4.3mL/min/kg.

    Description

    Clonidine is an imidazole derivate that acts as an agonist of alpha-2 adrenoceptors. This activity is useful for the treatment of hypertension, severe pain, and ADHD. Clonidine was granted FDA approval on 3 September 1974.

    Mechanism of Action

    Clonidine is primarily an alpha-2 adrenoceptor agonist which causes central hypotensive and anti-arrhythmogenic effects. The alpha-2 adrenoceptor is coupled to the G-proteins Go and Gi. Gi inhibits adenylyl cyclase and activates opening of a potassium channel that causes hyperpolarization. Clonidine binding to the alpha-2 adrenoceptor causes structural changes in the alpha subunit of the G-protein, reducing its affinity for GDP. Magnesium catalyzes the replacement of GDP with GTP. The alpha subunit dissociates from the other subunits and associates with an effector. The stimulation of alpha-2 adrenoceptors in the locus coeruleus may be responsible for the hypnotic effects of clonidine as this region of the brain helps regulate wakefulness. Clonidine can also decrease transmission of pain signals at the spine. Finally clonidine can affect regulators of blood pressure in the ventromedial and rostral-ventrolateral areas of the medulla.

    Pharmacodynamics

    Clonidine functions through agonism of alpha-2 adrenoceptors which have effects such as lowering blood pressure, sedation, and hyperpolarization of nerves. It has a long duration of action as it is given twice daily and the therapeutic window is between 0.1mg and 2.4mg daily.

    Metabolism

    The metabolism of clonidine is poorly understood. The main reaction in clonidine metabolism is the 4-hydroxylation of clonidine by CYP2D6, CYP1A2, CYP3A4, CYP1A1, and CYP3A5. Clonidine is <50% metabolized in the liver to inactive metabolites.

    Absorption

    Clonidine reaches maximum concentration in 60-90 minutes after oral administration. Race and fasting status do not influence pharmacokinetics of clonidine. A 100µg oral clonidine tablet reaches a Cmax of 400.72pg/mL with an AUC of 5606.78h\*pg/mL and a bioavailability of 55-87%.

    Toxicity

    Oral LD50 is 126 mg/kg in rats. The TDLO is 70µg/kg in children, 126µg/kg in women, and 69µg/kg in men. Symptoms of overdose include hypertension followed by hypotension, bradycardia, respiratory depression, hypothermia, drowsiness, decreased reflexes, weakness, irritability, and miosis. Severe overdoses can cause reversible cardiac conduction defects or dysrhythmias, apnea, coma, and seizures. Induction of vomiting is not recommended due to CNS depression but gastric lavage or activated charcoal may be useful in recent ingestion. Dialysis is also unlikely to be beneficial. Overdose can be treated with supportive measures such as atropine sulfate for bradycardia, intravenous fluids or vasopressors for hypotension, vasodilators for hypertension, naloxone for respiratory depression, and blood pressure monitoring.

    Indication

    Clonidine tablets and transdermal systems are indicated for the treatment of hypertension alone or in combination with other medications. A clonidine injection is indicated for use with opiates in the treatment of severe cancer pain where opiates alone are insufficient. An extended release tablet of clonidine is indicated for the treatment of ADHD either alone or in combination with other medications. Clonidine is also used for the diagnosis of pheochromocytoma, treatment of nicotine dependance, and opiate withdrawal.

    Half-life

    The elimination half life after epidural administration is 30 minutes but otherwise can range from 6-23h.

    Protein Binding

    Clonidine is 20-40% bound to plasma proteins, especially albumin.

    Elimination

    Approximately 50% of a clonidine dose is excreted in the urine as the unchanged drug and 20% is eliminated in the feces.

    Volume of Distribution

    The volume of distribution of clonidine has been reported as 1.7-2.5L/kg, 2.9L/kg, or 2.1±0.4L/kg depending on the source.

    Tolerance & Pharmacokinetics

    drugs.wiki

    Tolerance Decay

    Full tolerance 3d Half tolerance 8d Baseline ~14d

    Experience Report Analysis

    Erowid
    25 Reports
    1993–2020 Date Range
    6 With Age Data
    9 Effects Detected

    Demographics

    Gender Distribution

    Age Distribution

    Reports Over Time

    Effect Analysis

    Erowid

    Effects aggregated from 25 experience reports (25 Erowid)

    25 Reports
    9 Effects Detected
    6 Positive
    2 Adverse
    1 Neutral

    Effect Sentiment Distribution

    Confidence Distribution

    Positive Effects 6

    Sedation 44.0% 70%
    Stimulation 28.0% 70%
    Anxiety Suppression 24.0% 70%
    Euphoria 20.0% 70%
    Focus Enhancement 12.0% 70%
    Music Enhancement 12.0% 70%

    Adverse Effects 2

    Increased Heart Rate 16.0% 70%
    Motor Impairment 12.0% 70%

    Dosage Distribution

    Dose distribution from experience reports

    Median: 0.3 mg IQR: 0.2–0.9 mg n=10

    Real-World Dose Distribution

    62K Doses

    From 31 individual dose entries

    Oral (n=26)

    Median: 0.23mg 25th: 0.1mg 75th: 0.52mg 90th: 1.2mg
    mg/kg median: 0.003 mg/kg 75th: 0.006

    Form / Preparation

    Most common forms and preparations reported

    Body-Weight Dosing

    Dose relative to body weight from reports with weight data

    Median: 0.005 mg/kg IQR: 0.002–0.012 mg/kg n=10

    Redose Patterns

    Redosing behavior across 20 reports

    0.0% Redosed
    1.0 Avg Doses
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