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    DOM molecular structure

    DOM Stats & Data

    Stp And peace Tranquility Stp (serenity Serenity, tranquility, and peace
    NPS DataHub
    MW245.75
    FormulaC12H20ClNO2
    CAS15589-00-1
    SMILES[Cl-].COc1cc(CC(C)N)c(OC)cc1C.[H+]
    InChIKeyIPIMRNFGYPQKAM-UHFFFAOYSA-N
    Phenethylamines; 2020/1. Von 2-Phenethylamin abgeleitete Verbindungen; 2021/1. Von 2-Phenethylamin abgeleitete Verbindungen; 2022/1. Von 2-Phenethylamin abgeleitete Verbindungen
    Chemical Class Amphetamine
    Psychoactive Class Psychedelic / Stimulant
    Half-Life Unknown in humans; active effects are long (14–24 h). Metabolism involves CYP2D6 with hydroxy-DOM detectable by GC/MS in urine in animal models; poor metabolizers or CYP2D6 inhibition may prolong effects.

    Pharmacology

    DrugBank
    State Solid

    Description

    A psychedelic phenyl isopropylamine derivative, commonly called DOM, whose mood-altering effects and mechanism of action may be similar to those of LSD.

    Receptor Profile

    Receptor Actions

    Agonists
    5-HT2A receptor agonist (partial)
    5-HT2B receptor agonist (partial)
    5-HT2C receptor agonist (partial)

    History & Culture

    1963–1964

    DOM was first synthesized by Alexander Shulgin in 1963. The initial step of the synthesis was performed by his then fifteen-year-old son Theodore "Ted" Shulgin at Dow Chemical Company on June 22, 1963, during a brief period when the younger Shulgin had developed an interest in chemistry. Alexander Shulgin completed the synthesis on November 30, 1963. The compound was initially assigned the code name K-61,082, with "DOM" itself being an acronym derived from the code name "des-oxy-methyl" that Shulgin coined for the substance. Shulgin first discovered the effects of DOM on January 4, 1964, when he ingested a 1 mg dose orally. The hallucinogenic properties of the compound were subsequently confirmed on February 3, 1964, when Shulgin's colleague Thornton W. Sargent ingested 2.3 mg and experienced its full psychedelic effects.

    1967–present

    In mid-1967, DOM appeared on the black market in the Haight-Ashbury district of San Francisco during the height of the counterculture movement. Tablets containing 20 mg, and later 10 mg, of DOM were distributed widely under the name "STP," which was said to stand for "Serenity, Tranquility, and Peace," though alternative interpretations such as "Super Terrific Psychedelic" and "Stop The Police" also circulated. The name was actually borrowed from a motor oil additive that bore no chemical relation to the drug. These tablets were manufactured by underground chemists Owsley Stanley and Tim Scully. This distribution proved disastrous for several reasons. The tablets contained excessively high doses of the compound, and DOM's slow onset of action created particular problems for users accustomed to LSD. While LSD typically produces noticeable effects within fifteen to twenty minutes, DOM may only show initial effects at the half-hour mark, with full intensity not reached until approximately two hours after ingestion. Many users, expecting the rapid onset they knew from LSD and observing little activity after thirty minutes, assumed the dose was weak and consumed one or two additional tablets. This resulted in numerous hospitalizations, with treatment further complicated by the fact that medical personnel were initially unaware that the tablets called "STP" contained DOM. The negative publicity and health emergencies caused the supply of DOM to fade thereafter.

    Despite the troubled circumstances of its initial distribution, DOM holds a significant place in psychedelic history. Shulgin later documented the compound in his book PiHKAL and included it among his "magical half-dozen," a personal selection of the phenethylamine compounds he considered most important. This group comprises mescaline, DOM, 2C-B, 2C-E, 2C-T-2, and 2C-T-7, with DOM being one of the five that Shulgin himself developed and synthesized. There are reports that low doses of DOM were used as a stimulant by members of the Grateful Dead, which may represent one of the earliest documented instances of psychedelic microdosing.

    Subjective Effect Notes

    physical: The physical effects of DOM can be broken down into several components which progressively intensify proportional to dosage.

    cognitive: The cognitive effects of DOM are described by many as a combination of extreme mental stimulation and a powerful enhancement of a person's current mental state.

    Effect Profile

    Curated + 67 Reports
    Psychedelic 8.1

    Strong visuals, body load, headspace, and auditory effects

    Visual Intensity×3
    10104.5
    Headspace Depth×3
    96.73.6
    Auditory Effects×1
    8104.6
    Body Load / Somatic Effects×1
    10104.6
    Catalog Erowid BlueLight
    Stimulant 7.2

    Strong stimulation and anxiety/jitters with moderate euphoria and focus

    Stimulation / Energy×3
    1010
    Euphoria / Mood Lift×2
    76.4
    Focus / Productivity×2
    76.7
    Anxiety / Jitters×1
    1010
    Catalog Erowid

    Tolerance & Pharmacokinetics

    drugs.wiki
    Half-Life
    Unknown in humans; active effects are long (14–24 h). Metabolism involves CYP2D6 with hydroxy-DOM detectable by GC/MS in urine in animal models; poor metabolizers or CYP2D6 inhibition may prolong effects.
    Addiction Potential
    DOM is not considered physically addictive, but psychological reinforcement is possible. As with other classic psychedelics, frequent dosing rapidly builds tolerance and tends to diminish effects rather than escalate euphoric reinforcement.

    Tolerance Decay

    Full tolerance 1d Half tolerance 7d Baseline ~14d

    Acute tolerance rises rapidly after a single dose and can persist for days; functional tolerance commonly makes redosing ineffective and mostly prolongs duration. Cross-tolerance with other classic serotonergic psychedelics is expected. Data are primarily from community reports and general psychedelic pharmacology rather than controlled trials.

    Cross-Tolerances

    LSD
    70% ●○○
    Psilocybin
    60% ●○○
    Mescaline/2C-x
    60% ●○○

    Experience Report Analysis

    Erowid BlueLight
    54 Reports
    2000–2025 Date Range
    33 With Age Data
    30 Effects Detected

    Demographics

    Gender Distribution

    Age Distribution

    Reports Over Time

    Effect Analysis

    Erowid + Bluelight

    Effects aggregated from 67 experience reports (54 Erowid + 13 Bluelight)

    67 Reports
    96 Effects Detected
    36 Positive
    42 Adverse
    18 Neutral

    Effect Sentiment Distribution

    Confidence Distribution

    Positive Effects 36

    Stimulation 68.6% 83%
    Color Enhancement 58.2% 85%
    Music Enhancement 52.3% 85%
    Empathy 41.8% 80%
    Euphoria 41.8% 87%
    Focus Enhancement 33.3% 70%
    Tactile Enhancement 31.3% 85%
    Patterning 30.8% 85%
    Introspection 23.9% 79%
    Body High 20.9% 83%
    Serenity 15.4% 85%
    Mystical Quality 15.4% 85%
    Geometric Imagery 15.4% 92%
    Awe 7.7% 80%
    Fractal Imagery 7.7% 85%
    Vivid Dreams 7.7% 75%
    Love 7.7% 90%
    Oneness 7.7% 80%
    Shimmering 7.7% 85%
    Visual Trails 7.7% 85%

    Adverse Effects 42

    Anxiety 53.8% 84%
    Body Load 38.5% 88%
    Time Dilation 38.5% 79%
    Nausea 37.3% 85%
    Thought Disorganization 30.8% 80%
    Fear 23.1% 85%
    Vomiting 23.1% 83%
    Panic 23.1% 85%
    Sadness 23.1% 78%
    Insomnia 23.1% 82%
    Dysphoria 23.1% 82%
    Confusion 22.4% 75%
    Pupil Dilation 19.4% 87%
    Muscle Tension 16.4% 80%
    Dry Mouth 15.4% 85%
    Sweating 12.0% 85%
    Jaw Clenching 11.1% 70%
    Memory Suppression 9.3% 70%
    Increased Heart Rate 9.3% 70%
    Dizziness 7.7% 80%

    Dose-Response Correlation

    How effect frequency changes across dose levels

    View data table
    Effect Strong (n=12) Heavy (n=10)
    Visual Distortions 91.7% 70.0%
    Color Enhancement 83.3% 60.0%
    Stimulation 75.0% 80.0%
    Music Enhancement 66.7% 70.0%
    Euphoria 66.7% 40.0%
    Anxiety 66.7% 30.0%
    Empathy 58.3% 20.0%
    Confusion 33.3% 50.0%
    Tactile Enhancement 41.7% 20.0%
    Nausea 41.7% 40.0%
    Focus Enhancement 33.3% 40.0%
    Auditory Effects 25.0% 40.0%
    Sedation 33.3% 30.0%
    Closed-Eye Visuals 33.3% 20.0%
    Open-Eye Visuals 33.3% 30.0%

    Dose–Effect Mapping

    Experience Reports

    How reported effects shift across dose tiers, based on 54 experience reports.

    Limited tier coverage — most reports fall within the Strong / Heavy range. Effects at other dose levels may not be represented.

    Oral dose range: 5.0–11.3 mg (median 7.0 mg)
    Effect Strong (n=12) Heavy (n=10)
    visual distortions
    92%
    70%
    color enhancement
    83%
    60%
    stimulation
    75%
    80%
    music enhancement
    67%
    70%
    euphoria
    67%
    40%
    anxiety
    67%
    30%
    empathy
    58%
    20%
    confusion
    33%
    50%
    tactile enhancement
    42%
    20%
    nausea
    42%
    40%
    focus enhancement
    33%
    40%
    auditory effects
    25%
    40%
    sedation
    33%
    30%
    closed-eye visuals
    33%
    20%
    open-eye visuals
    33%
    30%
    muscle tension
    33%
    introspection
    17%
    20%
    hospital
    20%
    body high
    20%
    psychosis
    20%

    Showing top 20 of 23 effects

    Risk Escalation

    Sentiment Analysis

    Average frequency of positive vs adverse effects across dose tiers

    Strong n=12
    8 positive 55.2% 5 adverse 38.3%
    Heavy n=10
    9 positive 41.1% 4 adverse 35.0%
    View effect breakdown

    Adverse Effects

    Effect Strong (n=12) Heavy (n=10) Change
    Anxiety
    67%
    30%
    -55%
    Confusion
    33%
    50%
    +50%
    Nausea
    42%
    40%
    -4%
    Muscle Tension
    33%
    0%
    Psychosis
    20%
    0%
    Memory Suppression
    17%
    0%

    Positive Effects

    Effect Strong (n=12) Heavy (n=10) Change
    Color Enhancement
    83%
    60%
    -27%
    Stimulation
    75%
    80%
    6%
    Music Enhancement
    67%
    70%
    4%
    Euphoria
    67%
    40%
    -40%
    Empathy
    58%
    20%
    -65%
    Tactile Enhancement
    42%
    20%
    -52%
    Focus Enhancement
    33%
    40%
    +20%
    Introspection
    17%
    20%
    +19%
    Body High
    20%
    0%

    Dosage Distribution

    Dose distribution from experience reports

    Median: 7.0 mg IQR: 5.0–11.3 mg n=22

    Real-World Dose Distribution

    62K Doses

    From 62 individual dose entries

    Oral (n=34)

    Median: 6.0mg 25th: 5.0mg 75th: 10.0mg 90th: 14.1mg
    mg/kg median: 0.08 mg/kg 75th: 0.149

    Sublingual (n=9)

    Median: 2.5mg 25th: 2.5mg 75th: 5.0mg 90th: 6.0mg
    mg/kg median: 0.037 mg/kg 75th: 0.073

    Common Combinations

    Most co-occurring substances in experience reports

    Form / Preparation

    Most common forms and preparations reported

    Body-Weight Dosing

    Dose relative to body weight from reports with weight data

    Oral

    Median: 0.091 mg/kg IQR: 0.05–0.184 mg/kg n=21

    Sublingual

    Median: 0.04 mg/kg IQR: 0.032–0.074 mg/kg n=7

    Redose Patterns

    Redosing behavior across 43 reports

    18.6% Redosed
    1.2 Avg Doses

    Legal Status

    Country Status Notes
    Australia Illegal Prohibited under a blanket ban covering all substituted phenethylamines, which includes the entire DOx family of compounds.
    Austria Illegal Controlled under the SMG (Suchtmittelgesetz Österreich). Possession, production, and sale are prohibited.
    Belgium Schedule I Listed under HOOFDSTUK II of Belgian controlled substances legislation. Possession, distribution, and production are prohibited.
    Brazil Illegal Listed on Portaria SVS/MS nº 344 under the name 'STP'. Possession, production, and sale are prohibited.
    Canada Schedule I Controlled substance under the Controlled Drugs and Substances Act. Possession, trafficking, and production are criminal offenses.
    Czech Republic Schedule I Classified as a Schedule I controlled substance under national drug legislation.
    Germany Anlage I BtMG Controlled under Anlage I of the Betäubungsmittelgesetz (Narcotics Act) since April 15, 1971. Manufacturing, possession, import, export, buying, selling, procuring, or dispensing without a license is illegal.
    Italy Tabella I Listed in Tabella I of the 'Tabelle delle sostanze stupefacenti e psicotrope'. Possession, purchase, and sale are illegal.
    Latvia Schedule I Classified as a Schedule I controlled substance under Latvian drug control legislation.
    New Zealand Class A Controlled as a Class A substance, the most restrictive classification under New Zealand's Misuse of Drugs Act.
    Russia Schedule I Classified as a Schedule I substance in the Russian Federation. Buying, selling, or possessing without a license is illegal.
    Switzerland Verzeichnis D Specifically named as a controlled substance under Verzeichnis D of Swiss narcotics legislation.
    United Kingdom Class A Controlled under the Misuse of Drugs Act 1971. Buying, selling, or possessing without a license is prohibited.
    United States Schedule I Controlled under the Controlled Substances Act. Manufacturing, buying, possessing, or distributing without a DEA license is illegal.

    Harm Reduction

    drugs.wiki

    DOM has a slow onset and an extremely long duration; this historically led to non-fatal overdoses in the late 1960s when strong tablets were redosed prematurely. Redosing is discouraged, as intensity can increase significantly several hours after ingestion. Cardiovascular stimulation and peripheral vasoconstriction are commonly reported with DOx compounds; avoid combining with other stimulants or vasoconstrictors and avoid strenuous activity or hot environments; stay cool and sip fluids regularly. Lithium has repeatedly been associated with seizures and severe adverse reactions when combined with serotonergic psychedelics (e.g., LSD); by extension treat DOM + lithium as a dangerous combination. Metabolism involves CYP2D6; potent CYP2D6 inhibitors (fluoxetine, paroxetine, quinidine) may increase exposure, and DOM/related DOx can themselves inhibit CYP2D6—be cautious with co-medications that rely on this pathway. Immunoassay urine screens may return class-positive for ‘amphetamines’ with substituted amphetamines; targeted confirmation (e.g., GC/MS) can detect hydroxy-DOM and other metabolites, and detection windows vary—do not rely on specific ‘one week’ claims. Because DOM is potent at low milligram doses, use a 1 mg-accurate scale or volumetric dosing to reduce dosing error; avoid insufflation due to prolonged vasoconstriction and unpredictable intensity. Be prepared for insomnia during and after the experience; plan 24–36 hours of downtime. Where available, use reagent testing and consider lab drug checking; be aware that DOx has been sold on blotter or as other psychedelics—always wait at least 3 hours before making any dosing decisions.

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