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    Ketamine molecular structure

    Ketamine Stats & Data

    K Ket Jet Kitty 2-cxm kittens
    NPS DataHub
    MW237.73
    FormulaC13H16ClNO
    CAS6740-88-1
    IUPAC2-(2-chlorophenyl)-2-methylaminocyclohexan-1-one
    SMILESCNC1(CCCCC1=O)c1ccccc1Cl
    InChIKeyYQEZLKZALYSWHR-UHFFFAOYSA-N
    Arylcyclohexylamines; 2021/6. Von Arylcyclohexylamin abgeleitete Verbindungen; 2022/6. Von Arylcyclohexylamin abgeleitete Verbindungen
    Chemical Class Arylcyclohexylamine
    Psychoactive Class Dissociative

    Pharmacology

    DrugBank
    State Solid

    Description

    Ketamine is an NMDA receptor antagonist with a potent anesthetic effect. It was developed in 1963 as a replacement for phencyclidine (PCP) by Calvin Stevens at Parke Davis Laboratories. It started being used for veterinary purposes in Belgium and in 1964 was proven that compared to PCP, it produced minor hallucinogenic effects and shorter psychotomimetic effects. It was FDA approved in 1970, and from there, it has been used as an anesthetic for children or patients undergoing minor surgeries but mainly for veterinary purposes.

    Mechanism of Action

    Ketamine interacts with N-methyl-D-aspartate (NMDA) receptors, opioid receptors, monoaminergic receptors, muscarinic receptors and voltage sensitive Ca ion channels. Unlike other general anaesthetic agents, ketamine does not interact with GABA receptors.

    Pharmacodynamics

    Ketamine is a rapid-acting general anesthetic producing an anesthetic state characterized by profound analgesia, normal pharyngeal-laryngeal reflexes, normal or slightly enhanced skeletal muscle tone, cardiovascular and respiratory stimulation, and occasionally a transient and minimal respiratory depression. The anesthetic state produced by Ketamine has been termed as "dissociative anesthesia" in that it appears to selectively interrupt association pathways of the brain before producing somesthetic sensory blockade. It may selectively depress the thalamoneocortical system before significantly obtunding the more ancient cerebral centers and pathways (reticular-activating and limbic systems). Ketamine enhances descending inhibiting serotoninergic pathways and can exert antidepressive effects. These effects are seen in concentrations ten times lower than the needed concentration for anesthetic proposes. The effect of ketamine can be described as analgesic by the prevention of central sensitization in dorsal horn neurons as well as by the inhibition on the synthesis of nitric oxide. Ketamine can present cardiovascular changes and bronchodilatation.

    Metabolism

    Ketamine presents a mainly hepatic metabolism and its major metabolite is norketamine. The biotransformation of ketamine corresponds to N-dealkylation, hydroxylation of the cyclohexone ring, conjugation to glucuronic acid and dehydration of the hydroxylated metabolites for the formation of cyclohexene derivatives.

    Absorption

    Ketamine absorption is very rapid and the bioavailability is around 93%. After the first pass metabolism, only 17% of the administered dose is absorbed. It distributes very rapidly and presents a distribution half-life of 1.95 min. The Cmax levels at peak reach 0.75 mcg/ml in plasma and 0.2 mcg/ml in cerebrospinal fluid.

    Toxicity

    Preclinical studies related to the blocking of NMDA receptors have shown an increase in apoptosis in the developing brain which results in cognitive deficits when used for longer than 3 hours. Toxicity studies regarding carcinogenesis have not been performed. Regarding mutagenesis and fertility, ketamine showed to be clastogenic and to not have effects on fertility.

    Indication

    Ketamine is indicated as an anesthetic agent for recommended diagnostic and surgical procedures. If skeletal muscle relaxation is needed, it should be combined with a muscle relaxant. If the surgical procedure involves visceral pain, it should be supplemented with an agent that obtunds visceral pain. Ketamine can be used for induction of anesthesia prior other general anesthetic agents and as a supplement of low potency agents. Reports have indicated a potential use of ketamine as a therapeutic tool for the management of depression when administered in lower doses. These reports have increased the interest for ketamine in this area and several clinical trials are launched for this indication.

    Half-life

    The reported half-life in preclinical studies for ketamine is 186 min.

    Protein Binding

    The plasma protein binding of ketamine accounts for 53.5% of the administered dose.

    Elimination

    Pharmacokinetic studies have resulted in the recovery of 85-95% of the administered dose in urine mainly in the form of metabolites. Some other routes of elimination of ketamine are bile and feces. When administered intravenously the resultant recovery is distributed by 91% of the administered dose in urine and 3% in feces.

    Volume of Distribution

    The apparent volume of distribution of the central compartment and at steady-state are 371.3 ml/kg and 4060.3 ml/kg, respectively.

    Clearance

    The clearance rate of ketamine is high and of around 95 L/h/70kg.

    Metabolites

    Norketamine (active, primary metabolite)
    Dehydronorketamine (DHNK)
    (2R,6R)-hydroxynorketamine (active, implicated in antidepressant effects)
    Hydroxynorketamines (HNKs)
    Conjugated hydroxylated derivatives

    Receptor Profile

    Receptor Actions

    Agonists
    κ-opioid receptor agonist
    Dopamine D2 receptor agonist (partial)
    Sigma-1 receptor agonist
    Antagonists
    NMDA receptor antagonist (uncompetitive)
    5-HT2 receptor antagonist
    5-HT1A receptor antagonist
    Nicotinic acetylcholine receptor antagonist
    Inhibitors
    Norepinephrine reuptake inhibitor

    Receptor Binding

    NMDA (racemic) Ki = 119 nM
    NMDA (S-ketamine) Ki = 300 nM
    NMDA (R-ketamine) Ki = 1.4 μM
    GluN2A IC50 = 330 nM
    GluN2B IC50 = 310 nM
    GluN2C IC50 = 510 nM
    GluN2D IC50 = 830 nM
    D2 >10 μM (low affinity)
    α7 nAChR No significant binding (metabolites active)
    5-HT3A potentiator
    κ-opioid agonist
    μ-opioid binder (no agonist activity)
    δ-opioid binder
    NET inhibitor

    History & Culture

    Ketamine was first synthesized in 1962 by American scientist Calvin Stevens at Parke Davis Laboratories, initially designated "CI581." Stevens sought a safer anesthetic to replace phencyclidine, which produced severe and prolonged hallucinogenic effects upon recovery from anesthesia. By 1965, ketamine had demonstrated clinical utility as an anesthetic, and researcher Edward Domino coined the term "dissociative anesthetic" to describe its unique pharmacological profile. The United States Food and Drug Administration approved ketamine for human use in 1970. Ketamine's favorable safety margin proved particularly valuable in combat medicine. Its minimal respiratory depression compared to other available anesthetics, combined with its sympathomimetic properties, led to its extensive deployment as a field anesthetic during the Vietnam War. The substance remains on the World Health Organization's Essential Drugs List as one of the minimum medical requirements for a basic healthcare system.

    Recreational use of ketamine was first documented among medicinal chemists in the United States as early as 1965, spreading more widely by 1967. During the 1970s, patients recovering from ketamine anesthesia began reporting unwanted visions, drawing broader attention to the substance's psychoactive properties beyond clinical settings. In 1978, neuroscientist John Lilly published "The Scientist," an autobiographical account describing his extensive personal explorations with ketamine that helped popularize awareness of its dissociative effects among countercultural audiences. Recreational use expanded through the 1980s, and by the early 1990s ketamine had become more widespread in Europe, where it gained notoriety as an adulterant in MDMA tablets and became closely associated with the nightclub and rave scenes. By 1995, the United States Drug Enforcement Administration had placed ketamine on its emerging drugs list due to its growing presence in electronic music culture.

    Ketamine has accumulated numerous street names reflecting its diverse cultural contexts, including "Special K," "K," "Kit Kat," "kitty," "cat tranquilizer" (referencing its veterinary applications), "Cat Valium," and "Jet." The term "K-hole" describes a profound dissociative state involving visual and auditory hallucinations achieved at high doses, which has become a significant concept within psychedelic culture. Several prominent figures documented their entheogenic experiences with ketamine extensively, including John Lilly, Marcia Moore, and D.M. Turner. Turner's death by drowning during unsupervised ketamine use became a cautionary example of the serious physical risks posed by the drug's dissociative and anesthetic properties. More recently, actor Matthew Perry's death in October 2023, attributed primarily to the acute effects of ketamine, brought renewed public attention to the substance's dangers. Statistical data revealed more than 90 ketamine-related deaths in England and Wales between 2005 and 2013.

    The identification of ketamine's rapid antidepressant effects in 2000 represented a major breakthrough in psychiatric treatment. Research demonstrated that sub-anesthetic doses could produce immediate or near-immediate relief of depressive and suicidal symptoms, with therapeutic effects persisting up to three days following a single administration. This discovery has been characterized as the single most important advance in depression treatment in more than fifty years. The finding fundamentally shifted the direction of antidepressant research and development, generating widespread interest in NMDA receptor antagonists as a novel class of rapidly-acting treatments for mood disorders.

    Subjective Effect Notes

    physical: The subjective physical effects of ketamine can be broken down into eight components all of which progressively intensify proportional to dosage.

    cognitive: In comparison to other dissociatives, the cognitive effects of ketamine are often described as particularly forceful towards introspection and with more analytical thought process when compared to that of DXM and MXE.

    Effect Profile

    Curated + 920 Reports
    Dissociative 6.9

    Strong dissociative depth with moderate insight and motor impairment, low mania

    Dissociative Depth×3
    89.03.4 13/23
    Mania / Compulsion×1
    32.82.6 6/23
    Insight / Novel Thought×2
    70.72.0 0/23
    Motor / Sensory Impairment×1
    74.42.4 4/23
    Catalog Erowid BlueLight

    User Experiences

    Dissociative Depth "K is immensely bleak and the K Hole is just one great void." Effect Index
    Motor Impairment "I feel like I’ve snorted ice, that this icy numbness is radiating from my sinuses outwards." Effect Index
    Mania "Still quite dissociative but with a very "on the nose" euphoria." Bluelight

    Duration Timeline

    Bluelight
    Onset Comeup Peak Offset After Effects
    Oral
    10-30 minutes
    4-19 minutes
    45 minutes - 1.5 hours
    30 minutes - 1.0 hours
    4-8 hours
    Total: 1-2 hours
    Sublingual
    4-10 minutes
    4-19 minutes
    30 minutes
    30 minutes - 1.0 hours
    2-12 hours
    Total: 1-2 hours
    Insufflated
    4-15 minutes
    4-19 minutes
    30 minutes - 1.0 hours
    30 minutes - 1.0 hours
    2-12 hours
    Total: 1-2 hours
    Intramuscular
    1-4 minutes
    4-15 minutes
    15-45 minutes
    30 minutes - 1.0 hours
    2-12 hours
    Total: 1-2 hours
    Intravenous
    1-3 minutes
    3-4 minutes
    30 minutes - 1.0 hours
    2-12 hours
    Total: 1-2 hours
    Rectal
    10-19 minutes
    10-19 minutes
    30-40 minutes
    30 minutes - 1.0 hours
    2-12 hours
    Total: 1-2 hours

    Empirical Duration

    Erowid Reports
    Onset Come Up Peak Offset
    Insufflated (34 reports)

    Community Effects

    TripSit
    Positive
    hole
    Negative
    addiction

    Tolerance & Pharmacokinetics

    drugs.wiki

    Tolerance Decay

    Full tolerance 1d Half tolerance 21d Baseline ~28d

    Cross-Tolerances

    Dextromethorphan
    80% ●○○
    PCP
    80% ●○○
    Methoxetamine
    80% ●○○
    3-MeO-PCP
    80% ●○○
    3-HO-PCP
    80% ●○○
    3-MeO-PCE
    80% ●○○
    3-HO-PCE
    80% ●○○
    O-PCE
    80% ●○○

    Experience Report Analysis

    Erowid BlueLight
    712 Reports
    1994–2025 Date Range
    326 With Age Data
    33 Effects Detected

    Demographics

    Gender Distribution

    Age Distribution

    Reports Over Time

    Effect Analysis

    Erowid + Bluelight

    Effects aggregated from 762 experience reports (712 Erowid + 208 Bluelight)

    762 Reports
    153 Effects Detected
    54 Positive
    57 Adverse
    42 Neutral

    Effect Sentiment Distribution

    Confidence Distribution

    Positive Effects 54

    Music Enhancement 39.2% 83%
    Euphoria 32.7% 84%
    Color Enhancement 32.3% 82%
    Geometric Imagery 32.0% 86%
    Awe 30.0% 82%
    Empathy 29.6% 80%
    Stimulation 27.9% 83%
    Focus Enhancement 25.8% 70%
    Out-Of-Body Experience 24.0% 88%
    Tactile Enhancement 23.4% 80%
    Insight 22.0% 86%
    Mystical Quality 18.0% 85%
    Thought Acceleration 18.0% 79%
    Introspection 16.2% 80%
    Environmental Transfiguration 16.0% 87%
    Sociability Enhancement 14.0% 84%
    Lightness 12.0% 81%
    Contentment 12.0% 82%
    Oneness 12.0% 86%
    Empathogenic Connection 12.0% 80%

    Adverse Effects 57

    Anxiety 45.7% 83%
    Body Load 34.0% 81%
    Confusion 31.1% 87%
    Fear 28.0% 85%
    Nausea 19.6% 83%
    Motor Impairment 14.3% 89%
    Ataxia 14.0% 84%
    Memory Suppression 12.2% 80%
    Thought Disorganization 12.0% 81%
    Delusion 10.0% 80%
    Body Distortion 10.0% 83%
    Panic 10.0% 83%
    Paranoia 8.0% 78%
    Paranoid Ideation 6.0% 68%
    Communication Suppression 6.0% 80%
    Focus Suppression 6.0% 72%
    Pain Enhancement 6.0% 90%
    Appetite Suppression 6.0% 75%
    Amnesia 6.0% 85%
    Dysphoria 6.0% 85%

    Dose-Response Correlation

    How effect frequency changes across dose levels

    Insufflated

    View data table
    Effect Common (n=29) Strong (n=27) Heavy (n=67)
    Visual Distortions 79.3% 66.7% 58.2%
    Dissociation 62.1% 51.9% 71.6%
    Focus Enhancement 58.6% 25.9% 29.9%
    Anxiety 51.7% 55.6% 56.7%
    Music Enhancement 55.2% 44.4% 50.7%
    Closed-Eye Visuals 48.3% 25.9% 20.9%
    Color Enhancement 48.3% 40.7% 34.3%
    Confusion 48.3% 14.8% 37.3%
    Euphoria 34.5% 33.3% 46.3%
    Empathy 31.0% 44.4% 38.8%
    Auditory Effects 41.4% 22.2% 26.9%
    Nausea 37.9% 22.2% 23.9%
    Motor Impairment 37.9% 7.4% 9.0%
    Stimulation 27.6% 37.0% 26.9%
    Tactile Enhancement 20.7% 22.2% 28.4%

    Intramuscular

    View data table
    Effect Strong (n=16) Heavy (n=26)
    Visual Distortions 50.0% 73.1%
    Dissociation 50.0% 57.7%
    Anxiety 43.8% 57.7%
    Color Enhancement 56.2% 34.6%
    Music Enhancement 56.2% 50.0%
    Euphoria 50.0% 30.8%
    Empathy 43.8% 26.9%
    Focus Enhancement 31.2% 42.3%
    Auditory Effects 31.2% 38.5%
    Confusion 18.8% 38.5%
    Stimulation 37.5% 34.6%
    Nausea 37.5% 30.8%
    Tactile Enhancement 25.0% 26.9%
    Ego Dissolution 25.0% 15.4%
    Introspection 25.0% 19.2%

    Subjective Effect Ontology

    Experience Reports

    Structured effect tags extracted from 920 Erowid & Bluelight experience reports using a controlled vocabulary of 220+ canonical effects across 15 domains.

    Auditory

    music enhancement 299 34.2%

    Cognitive

    confusion 237 28.2% focus enhancement 184 25.8% introspection 123 20.3%

    Emotional

    anxiety 348 40.6% euphoria 249 32.2% empathy 225 24.8%

    Gastrointestinal

    nausea 149 18.3%

    Motor

    stimulation 212 24.1% sedation 111 12.4%

    Selfhood

    dissociation 387 43.4% ego dissolution 104 17.5%

    Tactile

    tactile enhancement 178 19.7%

    Visual

    visual distortions 336 37.2% color enhancement 246 29.5% closed eye visuals 154 24.0%

    16 unique effects extracted · Derived from Erowid & Bluelight reports

    Dose–Effect Mapping

    Experience Reports

    How reported effects shift across dose tiers, based on 712 experience reports.

    Insufflated dose range: 60.0–250.0 mg (median 150.0 mg)
    Effect Common (n=29) Strong (n=27) Heavy (n=67)
    visual distortions
    79%
    67%
    58%
    dissociation
    62%
    52%
    72%
    focus enhancement
    59%
    26%
    30%
    anxiety
    52%
    56%
    57%
    music enhancement
    55%
    44%
    51%
    closed-eye visuals
    48%
    26%
    21%
    color enhancement
    48%
    41%
    34%
    confusion
    48%
    15%
    37%
    euphoria
    34%
    33%
    46%
    empathy
    31%
    44%
    39%
    auditory effects
    41%
    22%
    27%
    nausea
    38%
    22%
    24%
    motor impairment
    38%
    7%
    9%
    stimulation
    28%
    37%
    27%
    tactile enhancement
    21%
    22%
    28%
    sedation
    24%
    26%
    19%
    muscle tension
    24%
    4%
    ego dissolution
    17%
    15%
    19%
    introspection
    17%
    18%
    19%
    body high
    10%
    15%
    19%

    Showing top 20 of 31 effects

    Intramuscular dose range: 50.0–125.0 mg (median 100.0 mg)
    Effect Strong (n=16) Heavy (n=26)
    visual distortions
    50%
    73%
    dissociation
    50%
    58%
    anxiety
    44%
    58%
    color enhancement
    56%
    35%
    music enhancement
    56%
    50%
    euphoria
    50%
    31%
    empathy
    44%
    27%
    focus enhancement
    31%
    42%
    auditory effects
    31%
    38%
    confusion
    19%
    38%
    stimulation
    38%
    35%
    nausea
    38%
    31%
    tactile enhancement
    25%
    27%
    ego dissolution
    25%
    15%
    introspection
    25%
    19%
    memory suppression
    12%
    23%
    sedation
    19%
    hospital
    19%
    12%
    motor impairment
    12%
    15%
    jaw clenching
    15%

    Showing top 20 of 25 effects

    Risk Escalation

    Sentiment Analysis

    Average frequency of positive vs adverse effects across dose tiers (Insufflated)

    Common n=29
    10 positive 31.0% 9 adverse 26.0%
    Strong n=27
    9 positive 31.2% 5 adverse 21.5%
    Heavy n=67
    10 positive 29.9% 11 adverse 15.1%
    View effect breakdown

    Adverse Effects

    Effect Common (n=29) Strong (n=27) Heavy (n=67) Change
    Anxiety
    52%
    56%
    57%
    9%
    Confusion
    48%
    15%
    37%
    -22%
    Nausea
    38%
    22%
    24%
    -36%
    Motor Impairment
    38%
    7%
    9%
    -76%
    Muscle Tension
    24%
    4%
    -81%
    Memory Suppression
    10%
    12%
    +15%
    Jaw Clenching
    10%
    7%
    6%
    -41%
    Sweating
    7%
    0%
    Increased Heart Rate
    7%
    3%
    -56%
    Headache
    6%
    0%
    Psychosis
    4%
    0%
    Seizure
    3%
    0%

    Positive Effects

    Effect Common (n=29) Strong (n=27) Heavy (n=67) Change
    Focus Enhancement
    59%
    26%
    30%
    -48%
    Music Enhancement
    55%
    44%
    51%
    -8%
    Color Enhancement
    48%
    41%
    34%
    -28%
    Euphoria
    34%
    33%
    46%
    +34%
    Empathy
    31%
    44%
    39%
    +25%
    Stimulation
    28%
    37%
    27%
    -2%
    Tactile Enhancement
    21%
    22%
    28%
    +37%
    Introspection
    17%
    18%
    19%
    +12%
    Body High
    10%
    15%
    19%
    +88%
    Creativity Enhancement
    7%
    4%
    -34%

    Risk Escalation

    Sentiment Analysis

    Average frequency of positive vs adverse effects across dose tiers (Intramuscular)

    Strong n=16
    8 positive 40.6% 6 adverse 22.9%
    Heavy n=26
    9 positive 30.8% 7 adverse 26.9%
    View effect breakdown

    Adverse Effects

    Effect Strong (n=16) Heavy (n=26) Change
    Anxiety
    44%
    58%
    +31%
    Confusion
    19%
    38%
    +104%
    Nausea
    38%
    31%
    -17%
    Memory Suppression
    12%
    23%
    +84%
    Motor Impairment
    12%
    15%
    +23%
    Jaw Clenching
    15%
    0%
    Muscle Tension
    12%
    0%
    Psychosis
    8%
    0%

    Positive Effects

    Effect Strong (n=16) Heavy (n=26) Change
    Color Enhancement
    56%
    35%
    -38%
    Music Enhancement
    56%
    50%
    -11%
    Euphoria
    50%
    31%
    -38%
    Empathy
    44%
    27%
    -38%
    Focus Enhancement
    31%
    42%
    +35%
    Stimulation
    38%
    35%
    -7%
    Tactile Enhancement
    25%
    27%
    7%
    Introspection
    25%
    19%
    -23%
    Body High
    12%
    0%

    Dosage Distribution

    Dose distribution from experience reports

    Oral

    Median: 320.0 mg IQR: 100.0–650.0 mg n=15

    Insufflated

    Median: 150.0 mg IQR: 60.0–250.0 mg n=131

    Intramuscular

    Median: 100.0 mg IQR: 50.0–125.0 mg n=54

    Intravenous

    Median: 50.0 mg IQR: 50.0–111.7 mg n=11

    Real-World Dose Distribution

    62K Doses

    From 830 individual dose entries

    Rectal (n=32)

    Median: 100.0mg 25th: 71.25mg 75th: 110.0mg 90th: 140.0mg
    mg/kg median: 2.05 mg/kg 75th: 3.646

    Insufflated (n=297)

    Median: 90.0mg 25th: 40.0mg 75th: 180.0mg 90th: 300.0mg
    mg/kg median: 1.192 mg/kg 75th: 2.671

    Oral (n=29)

    Median: 240.0mg 25th: 100.0mg 75th: 375.0mg 90th: 664.0mg
    mg/kg median: 1.837 mg/kg 75th: 6.369

    Intravenous (n=29)

    Median: 50.0mg 25th: 45.0mg 75th: 100.0mg 90th: 102.34mg
    mg/kg median: 0.814 mg/kg 75th: 1.548

    Intramuscular (n=124)

    Median: 70.0mg 25th: 50.0mg 75th: 110.0mg 90th: 147.0mg
    mg/kg median: 0.94 mg/kg 75th: 1.57

    Smoked (n=7)

    Median: 70.0mg 25th: 40.0mg 75th: 275.0mg 90th: 580.0mg
    mg/kg median: 1.029 mg/kg 75th: 3.982

    Sublingual (n=5)

    Median: 300.0mg 25th: 75.0mg 75th: 450.0mg 90th: 540.0mg

    Common Combinations

    Most co-occurring substances in experience reports

    Form / Preparation

    Most common forms and preparations reported

    Body-Weight Dosing

    Dose relative to body weight from reports with weight data

    Unknown

    Median: 2.206 mg/kg IQR: 0.882–3.049 mg/kg n=8

    Oral

    Median: 4.0 mg/kg IQR: 1.429–9.511 mg/kg n=12

    Insufflated

    Median: 2.195 mg/kg IQR: 0.862–3.529 mg/kg n=124

    Intramuscular

    Median: 1.192 mg/kg IQR: 0.667–1.778 mg/kg n=50

    Intravenous

    Median: 0.812 mg/kg IQR: 0.551–1.577 mg/kg n=12

    Redose Patterns

    Redosing behavior across 551 reports

    24.3% Redosed
    1.4 Avg Doses
    21m Median Interval

    Legal Status

    Not under international control (WHO Expert Committee on Drug Dependence recommended against scheduling in January 2016) WHO Essential Medicines List
    Country Status Notes
    Australia Schedule 8 Possession, manufacture, or supply without authority is illegal. Classified as a controlled drug requiring strict regulation.
    Austria Prescription only (NR medication) Legal for medical and veterinary use. Repeated dispense prohibited. Illegal when sold, possessed, or produced without a prescription under the Neue-Psychoaktive-Substanzen-Gesetz (NPSG).
    Belgium Prescription only Legal for medical and veterinary use. Possession without prescription can result in arrest.
    Brazil Controlled (veterinary) Legal for veterinary use only. Illegal when sold or possessed for human use.
    Cambodia Controlled Not legal to possess without a prescription. Some pharmacies may sell ketamine, though most consider it forbidden.
    Canada Schedule I (CDSA) Controlled under the Controlled Drugs and Substances Act as an analogue of phencyclidine (PCP). Sale, possession, and production are illegal unless authorized for medical, scientific, or industrial purposes.
    China Schedule II Classified as a class II psychiatric drug. Trade is limited to licensed wholesalers and retail sales are prohibited.
    Croatia Controlled Possession and distribution regulated under national drug legislation.
    Czech Republic Schedule IV (List 7) Available exclusively with a prescription under Nařízení vlády č. 463/2013 Sb. Sales and distribution controlled under the Medicines Act.
    Denmark Controlled Illegal except for scientific and medicinal uses as of February 8, 2008.
    France Schedule IV (Narcotic) Listed as a narcotic in Annexe IV of the Liste des substances classées comme stupéfiants. Injectable preparations classified as Médicine - List I and subject to strict controls.
    Germany Prescription only (Anlage 1 AMVV) Available by prescription for medical and veterinary use. Illegal when sold or possessed without a prescription.
    Hong Kong Schedule I Classified as a dangerous drug with strict controls on possession, manufacture, and distribution.
    India Unscheduled Not listed under the Narcotic Drugs and Psychotropic Substances Act, 1985. Reportedly available over-the-counter in many pharmacies.
    Italy Tabella I Listed in Tabella I of Tabelle delle sostanze stupefacenti e psicotrope. Illegal to possess, purchase, or sell.
    Japan Narcotic Classified as a narcotic under the Narcotic and Psychotropic Drugs Control Act (麻薬及び向精神薬取締法).
    Luxembourg Prohibited Prohibited substance for recreational use under national drug legislation.
    Malaysia Illegal Controlled under Section 39b of the Dangerous Drugs Act. Possession and sale are illegal.
    Mexico Category 3 Classified under Mexico's General Health Law as having therapeutic value but constituting a problem for public health. Acquisition restricted to licensed veterinarians only under Medicines Administration Regulations.
    Netherlands Unscheduled (medication) Treated as a medication rather than a controlled substance. Not listed in List 1 or 2 of the Opium Act. Sales require appropriate pharmaceutical licensing.
    New Zealand Class C Reclassified as a Class C substance in February 2008, the same category as cannabis and codeine.
    Norway Class A Classified as a Class A drug since 1999. Sold under the brand name Ketalar for authorized medical use.
    Poland Controlled Illegal to possess, manufacture, and sell except for authorized medical purposes.
    Singapore Class A Class A controlled drug. Traffickers face 5-20 years imprisonment and 5-15 strokes of the cane. Possession of more than 113g is deemed trafficking.
    Slovakia Schedule II Added to Schedule II (Grade 2) in October 2009, aligned with the 1971 United Nations Convention on Psychotropic Substances.
    South Korea Controlled Controlled substance as of January 2005. Possession and sale are illegal.
    Spain Schedule IV Classified as a Schedule IV substance under Spanish drug control legislation.
    Sweden Schedule IV Scheduled as of July 1, 2005 following increased reports of recreational misuse.
    Switzerland Controlled (Verzeichnis B) Specifically named under Verzeichnis B. Possession or handling without a license is illegal. Medicinal use is permitted.
    Taiwan Schedule III Controlled under national narcotics legislation with restrictions on possession and distribution.
    Turkey Green prescription only Available only with a 'green prescription' designation. Illegal when sold or possessed without a prescription.
    United Kingdom Class B Reclassified as a Class B drug under the Misuse of Drugs Act 1971 on June 10, 2014. Previously classified as Class C from January 1, 2006. Prior to 2006, possession was legal though sales required licensing.
    United States Schedule III Emergency scheduled by the DEA on August 12, 1999. Previously unscheduled with FDA-regulated sales. Possession without prescription or license is illegal. Esketamine nasal spray (Spravato) was FDA-approved in March 2019 for treatment-resistant depression under restricted distribution.
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