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    1cP-MiPLA molecular structure

    1cP-MiPLA Stats & Data

    PubChem
    MW391.5
    FormulaC24H29N3O2
    LogP3.3
    IUPAC(6aR,9R)-4-(cyclopropanecarbonyl)-N,7-dimethyl-N-propan-2-yl-6,6a,8,9-tetrahydroindolo[4,3-fg]quinoline-9-carboxamide
    InChIKeyDBIYDHUGEKMIHZ-DYESRHJHSA-N
    Chemical Class Lysergamide
    Psychoactive Class Psychedelic
    Half-Life Parent 1‑acyl compound likely hydrolyzes rapidly (minutes–<1 h, by analogy to 1‑acyl LSDs); active MiPLA human t\u0000bd unknown—assume on the order of a few hours similar to LSD; treat as estimate.

    Pharmacology

    DrugBank

    Receptor Profile

    Receptor Actions

    Agonists
    5-HT2A receptor agonist (partial)
    Other
    prodrug (metabolizes to MiPLA)

    History & Culture

    1cP-MiPLA is a novel synthetic lysergamide that first emerged on online research chemical markets around 2020. The compound's origins remain somewhat unclear, with unverified reports attributing its initial synthesis to either Skyler Ulrich or a chemist or group operating under the name Gerstmann. It appeared alongside several other novel lysergamide derivatives, including 1cP-AL-LAD, as part of a wave of new designer psychedelics entering the grey market during this period. By 2022, the compound had been identified in Japan, indicating its spread beyond initial Western markets. Like many contemporary research chemicals, 1cP-MiPLA was developed and distributed primarily through online vendors catering to the research chemical community, with limited formal scientific investigation into its properties beyond forensic identification studies.

    Toxicity

    PsychonautWiki

    The toxicity and long-term health effects of recreational 1cP-MiPLA use do not seem to have been studied in any scientific context and the exact toxic dose is unknown. This is because 1cP-MiPLA is a research chemical with very little history of human usage. The body of anecdotal reports suggests that there are no negative health effects attributed to simply trying the substance by itself at low to moderate doses and using it very sparingly (but nothing can be completely guaranteed). [https://www.google.com/ Independent research] should always be done to ensure that a combination of two or more substances is safe before consumption. As with other psychedelic substances, there are relatively few physical side effects that have been reported associated with acute 1cP-MiPLA exposure.

    Overdose

    The LD50 of 1cP-MiPLA is unknown. Adverse psychological reactions are common especially at higher dosages. Some of these include anxiety, delusions, panic attacks and more rarely seizures.

    Addiction & dependence

    Although no formal studies have been conducted, it is not unreasonable to assume that as with LSD itself, 1cP-MiPLA is not habit-forming and that the desire to use it can actually decrease with use. Tolerance to the effects of 1cP-MiPLA is built almost immediately after ingestion. After that, it takes about 5-7 days for the tolerance to be reduced to half and 14 days to be back at baseline (in the absence of further consumption).

    Effect Profile

    Curated
    Psychedelic 7.5

    Strong visuals and auditory effects with moderate headspace and body load

    Visual Intensity×3
    10
    Headspace Depth×3
    6
    Auditory Effects×1
    8
    Body Load / Somatic Effects×1
    6

    Tolerance & Pharmacokinetics

    drugs.wiki
    Half-Life
    Parent 1‑acyl compound likely hydrolyzes rapidly (minutes–<1 h, by analogy to 1‑acyl LSDs); active MiPLA human t\u0000bd unknown—assume on the order of a few hours similar to LSD; treat as estimate.
    Addiction Potential
    Low. Lysergamides do not produce physical dependence; compulsion/redosing is uncommon. Psychological craving is possible with frequent use or in specific contexts.

    Tolerance Decay

    Full tolerance 6h Half tolerance 5d Baseline ~14d

    Functional tolerance to lysergamides rises rapidly within a single session and decays over 5–14 days; acute redosing beyond ~45–60 min is typically inefficient. Cross‑tolerance is expected across serotonergic psychedelics that act via 5‑HT2A.

    Cross-Tolerances

    LSD
    80% ●●●
    Psilocybin/psilocin
    60% ●●○
    Other serotonergic psychedelics
    50% ●●○

    Legal Status

    Country Status Notes
    Austria Unscheduled (NPSG may apply) Not specifically scheduled, but may fall under the Neue-Psychoaktive-Substanzen-Gesetz (New Psychoactive Substances Act) as an analogue of LSD.
    Germany Illegal Controlled substance as of July 2021. Possession, production, and distribution are prohibited.
    Russia Schedule I Classified as a schedule I controlled substance under Russian drug control legislation.
    Switzerland Controlled (Verzeichnis E) Listed as a defined derivative of Lysergic Acid under Verzeichnis E point 263 of the controlled substances ordinance. Exempt when used for scientific or industrial purposes.
    United States Unscheduled Not specifically scheduled under the Controlled Substances Act. However, may be prosecuted as an analogue of LSD under the Federal Analogue Act when intended for human consumption.

    Harm Reduction

    drugs.wiki

    1cP‑MiPLA is a 1‑cyclopropionyl‑substituted lysergamide; by analogy to 1‑acyl LSD analogues, it is expected to hydrolyze in vivo to MiPLA and act primarily as a prodrug. Direct human pharmacokinetics are uncharacterized; assume delayed onset vs. parent amide and plan set/setting accordingly. Potency appears materially lower than LSD and highly product‑dependent; multiple user datasets report weak or threshold effects at 200 \u0000b5g and full effects at 300–800 \u0000b0g, highlighting the need to start low and titrate. Reagent tests: Ehrlich/Hofmann can ‘rule‑in’ an indole/lysergamide but cannot confirm identity or dose; only LC/MS‑based drug checking can. Some market blotters labeled as 1cP‑MiPLA have tested impure (e.g., significant iso‑isomers and unidentified byproducts), potentially reducing expected potency and altering side‑effect profile. Lysergamides produce rapid, pronounced acute tolerance—redosing after the first hour generally adds side‑effects and duration more than intensity. Like LSD, risks include anxiety/panic, vasoconstriction, hyperthermia in strenuous settings, and insomnia; a sober sitter, hydration, and temperature management reduce complication risk. Avoid if you have a history of psychosis or bipolar I; defer use during pregnancy. Leave at least 14 days between serotonergic psychedelic sessions to minimize tolerance stacking and potential HPPD risk. Never drive or engage in hazardous activities until fully baseline (often the next day).

    References

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