Summary
CBNA (cannabinolic acid) is the carboxylated precursor of cannabinol (CBN), formed when THCA oxidizes. It is produced naturally during storage and aging of cannabis, or rapidly when heated above ~110°C. CBNA itself has minimal affinity for CB1/CB2 receptors and is regarded as non-intoxicating. Limited animal research suggests mild anti-inflammatory and analgesic activity; anecdotal human use points to gentle sedation similar to low-dose CBN. Pharmacokinetic and toxicological data are sparse due to scarcity on the recreational market.
Dose Information
Light
Common
Strong
Heavy
Onset, Duration & After-effects
| ROA | Onset | Peak | Offset | After Effects |
|---|---|---|---|---|
| Oral | 30-90 min | 1.5-3 hrs | 1-2 hrs | 12 hrs |
| Sublingual | 10-30 min | 1.5-3 hrs | 1-2 hrs | 12 hrs |
Tolerance
Build-up
develops over weeks of regular use via CB1 receptor downregulation
Reset
2–4 weeks for most users; heavy users may need longer
Effects
Positive
- Physical relaxation
- Subtle body warmth
- Mild analgesia
- Stimulation
Negative
- Dry mouth
- Increased heart rate
- Nausea
- Headaches
- Headache
- Sedation
Positive
- Anxiety suppression
- Minimal euphoria
- Wakefulness
- Motivation enhancement
- Focus enhancement
- Memory enhancement
- Cognitive euphoria
- Focus Enhancement
Negative
- Minimal cognitive impairment
- Anxiety
Positive
- Increased libido
- Increased music appreciation
- Increased Music Appreciation
- Color enhancement
Negative
- Light sensitivity
- Appetite suppression
- Dehydration
- Appetite Suppression
- Light-headedness
- Dulled perception