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    Selegiline molecular structure

    Selegiline Stats & Data

    Emsam Jumex Zelapar Anipryl Deprenyl
    NPS DataHub
    MW187.28
    FormulaC13H17N
    CAS14611-51-9
    IUPAC(''R'')-''N''-methyl-''N''-(1-pheny
    SMILESC#CCN(C)C(C)Cc1ccccc1
    InChIKeyMEZLKOACVSPNER-GFCCVEGCSA-N
    Phenethylamines; 2020/1. Von 2-Phenethylamin abgeleitete Verbindungen; 2021/1. Von 2-Phenethylamin abgeleitete Verbindungen; 2022/1. Von 2-Phenethylamin abgeleitete Verbindungen
    Chemical Class Amphetamine
    Psychoactive Class Stimulant

    Pharmacology

    DrugBank
    Half-life 1.2-2 hours Protein binding > 99.5% State Solid

    Description

    A selective, irreversible inhibitor of Type B monoamine oxidase. It is used in newly diagnosed patients with Parkinson's disease. It may slow progression of the clinical disease and delay the requirement for levodopa therapy. It also may be given with levodopa upon onset of disability. (From AMA Drug Evaluations Annual, 1994, p385) The compound without isomeric designation is Deprenyl.

    Mechanism of Action

    Although the mechanisms for selegiline's beneficial action in the treatment of Parkinson's disease are not fully understood, the selective, irreversible inhibition of monoamine oxidase type B (MAO-B) is thought to be of primary importance. MAO-B is involved in the oxidative deamination of dopamine in the brain. Selegiline binds to MAO-B within the nigrostriatal pathways in the central nervous system, thus blocking microsomal metabolism of dopamine and enhancing the dopaminergic activity in the substantial nigra. Selegiline may also increase dopaminergic activity through mechanisms other than inhibition of MAO-B. At higher doses, selegiline can also inhibit monozmine oxidase type A (MAO-A), allowing it to be used for the treatment of depression.

    Pharmacodynamics

    Dopamine is an essential chemical that occurs in many parts of the body. It is the premature degradation of dopamine that results in the symptoms of Parkinson's disease. Monoamine oxidase (MAO) is an enzyme which accelerates the breakdown of dopamine. Selegiline can prolong the effects of dopamine in the brain by preventing its breakdown through seletively blocking MAO-B. It also may prevent the removal of dopamine between nerve endings and enhance release of dopamine from nerve cells.

    Absorption

    Rapidly absorbed from the gastrointestinal tract.

    Toxicity

    LD50=63 mg/kg (rats, IV)

    Indication

    Monotherapy for initial treatment of Parkinson's disease, as well as an adjunct therapy in patients with a decreased response to levodopa/carbadopa. Also used for the palliative treatment of mild to moderate Alzheimer's disease and at higher doses, for the treatment of depression.

    Effect Profile

    Curated + 15 Reports
    Stimulant 5.6

    Strong focus with moderate anxiety/jitters, mild stimulation and euphoria

    Stimulation / Energy×3
    5
    Euphoria / Mood Lift×2
    4
    Focus / Productivity×2
    9
    Anxiety / Jitters×1
    6

    Tolerance & Pharmacokinetics

    drugs.wiki

    Tolerance Decay

    Full tolerance 1d Half tolerance 12d Baseline ~21d

    Experience Report Analysis

    Erowid
    15 Reports
    2001–2020 Date Range
    4 With Age Data
    8 Effects Detected

    Demographics

    Gender Distribution

    Age Distribution

    Reports Over Time

    Effect Analysis

    Erowid

    Effects aggregated from 15 experience reports (15 Erowid)

    15 Reports
    8 Effects Detected
    4 Positive
    2 Adverse
    2 Neutral

    Effect Sentiment Distribution

    Confidence Distribution

    Positive Effects 4

    Stimulation 53.3% 70%
    Euphoria 40.0% 70%
    Empathy 26.7% 70%
    Focus Enhancement 20.0% 70%

    Adverse Effects 2

    Anxiety 46.7% 70%
    Increased Heart Rate 20.0% 70%

    Common Combinations

    Most co-occurring substances in experience reports

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